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Profil bibliographique

Nora M. Kochupurakkal

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

14Publications signalées
278Citations signalées
4Affiliations récentes

Les institutions déclarées

Les domaines associés

Angiogenesis and VEGF in CancerAxon Guidance and Neuronal SignalingT-cell and B-cell ImmunologyOrgan Transplantation Techniques and OutcomesReproductive System and Pregnancy

Les publications récentes

Accès ouvert 2025 article OpenAlex

Neuropilin-2 functions as a coinhibitory receptor to regulate antigen-induced inflammation and allograft rejection

Johannes Wedel, Nora M. Kochupurakkal, Sek Won Kong, Sayantan Bose et autres

Coinhibitory receptors function as central modulators of the immune response to resolve T effector activation and/or to sustain immune homeostasis. Here, using humanized SCID mice, we found that neuropilin-2 (NRP2) is inducible on late effector and exhausted subsets of human CD4+ T …

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2 citations Journal of Clinical Investigation
Accès ouvert 2025 preprint OpenAlex

Neuropilin-2 functions as a co-inhibitory receptor to regulate antigen-induced inflammation and allograft rejection

Johannes Wedel, Nora M. Kochupurakkal, Sek Won Kong, Sayantan Bose et autres

Abstract Co-inhibitory receptors function as central modulators of the immune response to resolve T effector activation and/or to sustain immune homeostasis. Here, using humanized SCID mice, we found that NRP2 is inducible on late effector and exhausted subsets of human CD4 + …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2016 article OpenAlex

The intragraft microenvironment as a central determinant of chronic rejection or local immunoregulation/tolerance

Johannes Wedel, Hironao Nakayama, Nora M. Kochupurakkal, Josephine Koch et autres

PURPOSE OF REVIEW: Chronic rejection is associated with persistent mononuclear cell recruitment, endothelial activation and proliferation, local tissue hypoxia and related biology that enhance effector immune responses. In contrast, the tumor microenvironment elicits signals/factors that inhibit effector T cell responses and rather …

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10 citations Current Opinion in Organ Transplantation
Accès ouvert 2016 article OpenAlex

CD11c.DTR mice develop a fatal fulminant myocarditis after local or systemic treatment with diphtheria toxin

Linda Männ, Nora M. Kochupurakkal, Christian Martin, Eva Verjans et autres

To assess the role of alveolar macrophages (AMs) during a pulmonary Aspergillus fumigatus infection AMs were depleted by intratracheal application of diphtheria toxin (DTX) to transgenic CD11c.DTR mice prior to fungal infection. Unexpectedly, all CD11c.DTR mice treated with DTX died within 4-5 …

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27 citations European Journal of Immunology
Accès ouvert 2015 article OpenAlex

Regulation of mTOR Signaling by Semaphorin 3F-Neuropilin 2 Interactions In Vitro and In Vivo

Hironao Nakayama, Sarah Bruneau, Nora M. Kochupurakkal, Sílvia Coma et autres

Semaphorin 3F (SEMA3F) provides neuronal guidance cues via its ability to bind neuropilin 2 (NRP2) and Plexin A family molecules. Recent studies indicate that SEMA3F has biological effects in other cell types, however its mechanism(s) of function is poorly understood. Here, we …

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72 citations Scientific Reports
Accès ouvert 2015 article OpenAlex

Chronic allograft rejection

Johannes Wedel, Sarah Bruneau, Nora M. Kochupurakkal, Leo Boneschansker et autres

PURPOSE OF REVIEW: New developments suggest that the graft itself and molecules expressed within the graft microenvironment dictate the phenotype and evolution of chronic rejection. RECENT FINDINGS: Once ischemia-reperfusion injury, cellular and humoral immune responses target the microvasculature, the associated local tissue …

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26 citations Current Opinion in Organ Transplantation
2014 article OpenAlex

Novel Effects of Semaphorin 3F On the Regulation of Intracellular PI3K-Akt Signaling.

Hironao Nakayama, Sarah Bruneau, Nora M. Kochupurakkal, Michael Klagsbrun et autres

Class three semaphorins, well characterized as axon guidance molecules, act via neuropilin (NRP) and plexin family receptors and elicit signals, resulting in anti-migration and cytoskeleton collapse. Recently, we have demonstrated that a) Semaphorin 3F (SEMA3F) is potent to inhibit allograft rejection in …

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0 citations Transplantation
2014 article OpenAlex

T-Cell Specific Adapter Protein (TSAd) Mediates Immunoregulation in Alloimmunity.

Matthew M. Sheehan, Matthew Stack, Takeshi Seto, K. Liu et autres

An understanding of the alloimmune response and events leading to immunoregulation is required to optimize long-term allograft survival. TSAd, an SH2 domain containing intracellular adapter molecule, functions to elicit T cell receptor-mediated signals that result in effector T cell (Teff) cytokine production …

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0 citations Transplantation
2014 article OpenAlex

NRP1 in Effector and Regulatory CD4+ T Cell Populations.

E. Lee, Nora M. Kochupurakkal, Hironao Nakayama, Hyunggun Kim et autres

Background: Neuropilins (NRP1 and NRP2) serve as receptors for class 3 family semaphorins and were originally described as axonal guidance molecules. More recently, NRP1 has been found to be highly expressed on most CD4+Foxp3+ regulatory T cells (Treg) and to facilitate DC-Treg …

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0 citations Transplantation
2014 article OpenAlex

A Novel Immunomodulatory Function for Semaphorin3F and Neuropilin-2 in Allograft Rejection.

Nora M. Kochupurakkal, Hironao Nakayama, G. Demirci, K. Macleaod et autres

The class3 semaphorins (Sema3A-G) bind to Neuropilin/Plexin complexes on tumor cells and elicit regulatory signals, anti-migration and anti-proliferation. Recently, we found that Sema3F modulates PI-3K-Akt and MAPK signaling via neuropilin-2 (NRP-2), suggesting that this ligand-receptor interaction may be potent to inhibit T …

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0 citations Transplantation
Accès ouvert 2014 article OpenAlex

Blockade of the Programmed Death-1 (PD1) Pathway Undermines Potent Genetic Protection from Type 1 Diabetes

Nora M. Kochupurakkal, Annie J. Kruger, Sudipta Tripathi, Bing Mei Zhu et autres

AIMS/HYPOTHESIS: Inhibition of PD1-PDL1 signaling in NOD mice accelerates onset of type 1 diabetes implicating this pathway in suppressing the emergence of pancreatic beta cell reactive T-cells. However, the molecular mechanism by which PD1 signaling protects from type 1 diabetes is not …

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60 citations PLoS ONE
Accès ouvert 2013 article OpenAlex

B7h (ICOS-L) Maintains Tolerance at the Fetomaternal Interface

Leonardo Vidal Riella, Shirine Dada, Lola Chabtini, Brian D. Smith et autres

In a successful pregnancy, the semiallogeneic fetus is not rejected by the maternal immune system, which implies tolerance mechanisms protecting fetal tissues from maternal immune attack. Here we report that the ICOS-B7h costimulatory pathway plays a critical role in maintaining the equilibrium …

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35 citations American Journal Of Pathology

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