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Profil bibliographique

Heba M. Amer

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

6Publications signalées
34Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

SARS-CoV-2 and COVID-19 ResearchCOVID-19 Clinical Research StudiesInflammasome and immune disordersNeuroinflammation and Neurodegeneration MechanismsMicroRNA in disease regulation

Les publications récentes

Accès ouvert 2026 article OpenAlex

Caspase-11–mediated hyperinflammation impairs CD8+ T cell immunity and viral clearance in severe SARS-CoV-2 infection

Mostafa M. Eltobgy, Mohamed M. Shamseldin, Owen D. Whitham, Heba M. Amer et autres

Severe SARS-CoV-2 infection is characterized by lung hyperinflammation, impaired IFN responses, and defective T cell activation, yet the molecular drivers of these immune dysregulations remain incompletely understood. Caspase-11 (CASP11), a key effector of the noncanonical inflammasome, has been shown to mediate an …

us, Égypte, it, Ouganda (code pays fourni par la source)

0 citations JCI Insight
Accès ouvert 2026 preprint OpenAlex

Gelsolin protects mitochondria and regulates inflammation during Legionella pneumophila infection

Owen D Whitham, Mostafa Eltobgy, Mohamed M. Shamseldin, Asmaa Badr et autres

Abstract Legionella pneumophila ( L. pneumophila ) is the causative agent of Legionnaires’ disease, a severe bacterial pneumonia. Difficulty in diagnosing Legionnaires’ disease leads to an underreporting of cases and delayed treatment. Rapid-acting, broad-spectrum therapies are needed to treat pathology while avoiding …

us, Égypte (code pays fourni par la source)

0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 preprint OpenAlex

Caspase-11 Mediated Hyperinflammation Impairs CD8⁺ T Cell Immunity and Viral Clearance in Severe SARS-CoV-2 Infection

Mostafa M. Eltobgy, Mohamed Α. Shamseldin, Owen Whitham, Heba M. Amer et autres

Abstract Severe SARS-CoV-2 infection is characterized by lung hyperinflammation, impaired interferon responses, and defective T-cell activation, yet the molecular drivers of these immune dysregulations remain incompletely understood. Caspase-11 (CASP11), a key mediator of the non-canonical inflammasome, has been shown to mediate an …

us (code pays fourni par la source)

1 citation bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 article OpenAlex

Caspase-4/11 induces lung inflammation and dampens the humoral immune response in SARS-CoV2 infected mice 3644

Mohamed Α. Shamseldin, Mostafa M. Eltobgy, Heba M. Amer, Owen Witham et autres

Abstract Description SARS-CoV2 devastated the world in 2019, with over 774 million cases and around 7 million deaths. Our lab has previously reported the role Caspase-4/11 plays in the innate immune response against SARS-CoV2, associating it with exacerbated lung pathology and immunothrombosis, …

us (code pays fourni par la source)

0 citations The Journal of Immunology
Accès ouvert 2025 article OpenAlex

Development of SARS-CoV-2 as a viral vector: A novel intranasal bivalent vaccine for SARS-CoV-2 and RSV

Jiayu Xu, Michelle Chamblee, Fei Jiang, Mahesh KC et autres

Negative-sense RNA viruses have been widely used as viral vectors for vaccine delivery. However, little is known about coronaviruses as vectors for delivering vaccines. Here, we have developed safe SARS-CoV-2 Omicron JN.1-based live attenuated vaccine candidates by combining a mutation (D130A) in …

us (code pays fourni par la source)

3 citations Science Advances
Accès ouvert 2025 preprint OpenAlex

AI-based decoding of long covid cognitive impairments in mice using automated behavioral system and comparative transcriptomic analysis

Heba M. Amer, Mohamed M. Shamseldin, Sarah Faber, Mostafa M. Eltobgy et autres

Long COVID (LC) following SARS-CoV-2 infection affects millions of individuals world-wide and manifests with a variety of symptoms including cognitive dysfunction also known as "brain fog". This is characterized by difficulties in executive functions, planning, decision-making, working memory, impairments in complex attention, …

us (code pays fourni par la source)

0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2024 article OpenAlex

Microglia-targeted inhibition of miR-17 via mannose-coated lipid nanoparticles improves pathology and behavior in a mouse model of Alzheimer’s disease

Asmaa N. Badr, Kylene P. Daily, Mostafa M. Eltobgy, Shady Z. K. Estfanous et autres

Neuroinflammation and accumulation of Amyloid Beta (Aβ) accompanied by deterioration of special memory are hallmarks of Alzheimer's disease (AD). Effective preventative and treatment options for AD are still needed. Microglia in AD brains are characterized by elevated levels of microRNA-17 (miR-17), which …

us (code pays fourni par la source)

30 citations Brain Behavior and Immunity

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