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Profil bibliographique

Cláudio Gouveia Roque

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

20Publications signalées
395Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Amyotrophic Lateral Sclerosis ResearchRNA Research and SplicingNuclear Receptors and SignalingAlzheimer's disease research and treatmentsNeurogenesis and neuroplasticity mechanisms

Les publications récentes

Accès ouvert 2026 article OpenAlex

Distinct cellular phenotypes of language and executive decline in amyotrophic lateral sclerosis

Joana Petrescu, Cláudio Gouveia Roque, Christopher A. Jackson, Aidan C. Daly et autres

Cognitive manifestations, including impairments in language and executive functions, are seen in amyotrophic lateral sclerosis (ALS), but the underlying mechanisms remain unclear. We mapped prefrontal cortex regions from ALS patients by integrating spatial and single-nucleus transcriptomics in a cognitively stratified patient cohort. …

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0 citations Cell
Accès ouvert 2026 preprint OpenAlex

TDP-43 dysfunction leads to the accumulation of cryptic transposable element-derived exons, crypTEs, in iPSC derived neurons and ALS/FTD patient tissues

Isobel Bolger, Regina Shaw, Oliver H. Tam, Cláudio Gouveia Roque et autres

TDP-43 is an RNA and DNA binding protein that plays major roles in regulating RNA processing. In particular, TDP-43 dysfunction leads to the accumulation of cryptic splice isoforms that result from improperly spliced mRNAs. In addition to its role in regulating splicing, …

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1 citation bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2026 article OpenAlex

Reticulon-1 synthesis controls outgrowth and microtubule dynamics in injured cortical axons

Alejandro Luarte, Javiera Andrea Ojeda Gallardo, Daniela Corvalán, Ankush Chakraborty et autres

The regenerative potential of developing cortical axons depends on intrinsic mechanisms, such as axon-autonomous protein synthesis, that are still not fully understood. An emerging factor in this regenerative response is the bidirectional interplay between microtubule dynamics and the axonal ER. We hypothesize …

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0 citations Life Science Alliance
Accès ouvert 2025 article OpenAlex

High-resolution brain mapping in ALS: multiplexed insights into TDP-43 aggregation and disease mechanisms 2824

Zoé Butti, Christopher A. Jackson, Cláudio Gouveia Roque, Pedro Machado Almeida et autres

Abstract Description Abnormal protein aggregation is a central pathological feature of neurodegenerative diseases (PMID: 30258241), notably observed in Amyotrophic Lateral Sclerosis (ALS), with 97% of cases presenting TAR DNA-binding protein 43 (TDP-43) aggregation (PMID: 17469116). Cytoplasmic TDP-43 inclusions manifest not only in …

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0 citations The Journal of Immunology
Accès ouvert 2025 article OpenAlex

Alpha-synuclein abundance and localization are regulated by the RNA-binding protein PUMILIO1

Maximilian Cabaj, Pietro Giuseppe Mazzara, Zachary Gaertner, Ruizhi Wang et autres

The protein α-synuclein, encoded by SNCA, accumulates in Parkinson's disease (PD) and other synucleinopathies for reasons that remain unclear. Here, we investigated whether SNCA is regulated in vivo by the RNA-binding protein PUM1. We establish that PUM1 binds to SNCA's 3' UTR …

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6 citations Cell Reports
Accès ouvert 2025 preprint OpenAlex

FUS Mislocalization Rewires a Cortical Gene Network to Drive Cognitive and Behavioral Impairment in ALS

Raphaelle Cassel, Félicie Lorenc, Aurélie Bombardier, Claudia De Tapia et autres

Summary Cognitive and behavioral impairment affects up to half of individuals with amyotrophic lateral sclerosis (ALS), but their molecular origin remains unresolved. Here, we identify mislocalization of the RNA-binding protein FUS in cortical neurons as a defining feature in ALS patients with …

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3 citations medRxiv
Accès ouvert 2025 article OpenAlex

Modulation of CREB3L2-ATF4 heterodimerization via proteasome inhibition and HRI activation in Alzheimer’s disease pathology

Krystal Herline, Michaela M Pauers, Jessica E. Lipponen, Michael A. Zrzavy et autres

Abstract Alzheimer’s disease (AD) pathology includes transcriptional changes in the neurons, which are in part caused by the heterodimerization of two stress response transcription factors, CREB3L2 and ATF4. We investigated the role of proteasome inhibition and the eIF2α-kinase HRI in the formation …

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2 citations Cell Death and Disease
Accès ouvert 2025 preprint OpenAlex

Distinct Cellular Phenotypes of Language and Executive Decline in Amyotrophic Lateral Sclerosis

Joana Petrescu, Cláudio Gouveia Roque, Christopher A. Jackson, Aidan C. Daly et autres

Cognitive manifestations, including impairment in language and executive functions, are seen in amyotrophic lateral sclerosis (ALS), but the mechanisms that underlie these deficits remain unclear. To address this, we mapped prefrontal cortex regions from ALS patients by integrating spatial and single-nucleus transcriptomics …

us, gb, no (code pays fourni par la source)

3 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2024 article OpenAlex

The broken Alzheimer’s disease genome

Cláudio Gouveia Roque, Hemali Phatnani, Ulrich Hengst

The complex pathobiology of late-onset Alzheimer's disease (AD) poses significant challenges to therapeutic and preventative interventions. Despite these difficulties, genomics and related disciplines are allowing fundamental mechanistic insights to emerge with clarity, particularly with the introduction of high-resolution sequencing technologies. After all, …

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14 citations Cell Genomics
Accès ouvert 2023 article OpenAlex

CREB3L2-ATF4 heterodimerization defines a transcriptional hub of Alzheimer’s disease gene expression linked to neuropathology

Cláudio Gouveia Roque, Kyung Min Chung, Ethan P. McCurdy, Radhika Jagannathan et autres

Gene expression is changed by disease, but how these molecular responses arise and contribute to pathophysiology remains less understood. We discover that β-amyloid, a trigger of Alzheimer's disease (AD), promotes the formation of pathological CREB3L2-ATF4 transcription factor heterodimers in neurons. Through a …

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26 citations Science Advances
Accès ouvert 2022 article OpenAlex

A systemic cell stress signal confers neuronal resilience toward oxidative stress in a Hedgehog-dependent manner

Kyung Min Chung, Hyunha Kim, Cláudio Gouveia Roque, Ethan P. McCurdy et autres

Cells possess several conserved adaptive mechanisms to respond to stress. Stress signaling is initiated to reestablish cellular homeostasis, but its effects on the tissue or systemic levels are far less understood. We report that the secreted luminal domain of the endoplasmic reticulum …

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22 citations Cell Reports

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