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Profil bibliographique

Abel Martín-Garrido

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

22Publications signalées
482Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Cardiac Fibrosis and RemodelingCardiomyopathy and Myosin StudiesNitric Oxide and Endothelin EffectsCell Adhesion Molecules ResearchProtein Kinase Regulation and GTPase Signaling

Les publications récentes

Accès ouvert 2026 preprint OpenAlex

FYCO1 improves postischemic cardiac remodeling via enhanced autophagic flux and attenuation of proinflammatory signaling

Frauke Senger, Susanne S. Hille, Anca Kliesow Remes, Tapan Kumar Baral et autres

Abstract Acute myocardial infarction (MI) is associated with severe metabolic and oxidative stress that triggers cardiomyocyte death, pro-inflammatory signaling and progressive structural remodeling frequently culminating in heart failure. Although significant advances in reperfusion therapy improved acute survival in patients, therapeutic strategies that …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2026 preprint OpenAlex

Loss of cardiomyocyte eukaryotic elongation factor 1A2 in mice triggers cardiomyopathy due to defective proteostasis

Abel Martín-Garrido, Nadine Weyrauch, Jorge Ruiz‐Orera, Jeanette Eresch et autres

Abstract Eukaryotic elongation factor 1A (eEF1A) delivers aminoacyl-tRNAs to ribosomes but also has additional, non-canonical functions. Mammals express two paralogs: eEF1A1 is ubiquitous, whereas eEF1A2 is confined to adult cardiomyocytes, skeletal myocytes, and neurons. Mutations in EEF1A2 cause cardiomyopathy, but underlying mechanisms …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 erratum OpenAlex

Correction: Deficiency in nucleoside diphosphate kinase B leads to endothelial activation of the hexosamine biosynthesis pathway and cardiac dysfunction

Feng Shao, Johanna Wieland, Yixin Wang, Merve Keleş et autres

In the original publication of this article [1], the author’s name “Felix A. Trogisch” was incorrectly written as “Trogisch Felix”. The original article has been corrected.

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0 citations Cardiovascular Diabetology
2025 editorial OpenAlex

Cardiomyocyte-derived fibrosis as driver of cardiomyopathy

Abel Martín-Garrido, Joerg Heineke

This editorial refers to ‘Cardiomyocyte SORBS2 expression increases in heart failure and regulates integrin interactions and extracellular matrix composition’, by L.T. Timmer et al., https://doi.org/10.1093/cvr/cvaf021. Cardiac fibrosis describes pathological deposition of extracellular matrix (ECM), which can either be the consequence of cardiomyocyte …

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1 citation Cardiovascular Research
Accès ouvert 2025 article OpenAlex

Deficiency in nucleoside diphosphate kinase B leads to endothelial activation of the hexosamine biosynthesis pathway and cardiac dysfunction

Feng Shao, Johanna Wieland, Yixin Wang, Merve Keleş et autres

Abstract Background Nucleoside diphosphate kinase B (NDPKB) deficiency in endothelial cells (ECs) promotes the activation of the hexosamine biosynthesis pathway (HBP), leading to vascular damage in the retina. The aim of this study was to investigate the consequences of NDPKB deficiency in …

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6 citations Cardiovascular Diabetology
Accès ouvert 2024 article OpenAlex

Endothelial derived, secreted long non-coding RNAs Gadlor1 and Gadlor2 aggravate cardiac remodeling

Merve Keleş, Steve Grein, Natali Froese, Dagmar Wirth et autres

lncRNAs to cardiomyocytes, where they bind and activate calmodulin-dependent kinase II, and impact pro-hypertrophic gene expression and calcium homeostasis. Therefore, we reveal a crucial lncRNA-based mechanism of EC-cardiomyocyte crosstalk during heart failure, which could be specifically modified in the future for therapeutic …

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3 citations Molecular Therapy — Nucleic Acids
Accès ouvert 2024 article OpenAlex

Endothelial cells drive organ fibrosis in mice by inducing expression of the transcription factor SOX9

Felix A. Trogisch, Aya Abouissa, Merve Keleş, Anne Birke et autres

Fibrosis is a hallmark of chronic disease. Although fibroblasts are involved, it is unclear to what extent endothelial cells also might contribute. We detected increased expression of the transcription factor Sox9 in endothelial cells in several different mouse fibrosis models. These models …

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62 citations Science Translational Medicine
Accès ouvert 2023 article OpenAlex

Ythdf2 regulates cardiac remodeling through its mRNA target transcripts

Vivien Kmietczyk, J. Oelschläger, Parul Gupta, Eshita Varma et autres

m 6 A mRNA methylation controls cardiomyocyte function and increased overall m 6 A levels are a stereotyping finding in heart failure independent of the underlying etiology. However, it is largely unknown how the information is read by m 6 A reader …

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23 citations Journal of Molecular and Cellular Cardiology
Accès ouvert 2022 preprint OpenAlex

Endothelial cell-derived, secreted long non-coding RNAs Gadlor1 and Gadlor2 aggravate pathological cardiac remodeling via intercellular crosstalk

Merve Keleş, Steve Grein, Natali Froese, Dagmar Wirth et autres

Abstract Background Pathological overload triggers maladaptive myocardial remodeling that leads to heart failure. Recent studies have shown that long non-coding RNAs (lncRNAs) regulate cardiac remodeling. This study investigates two recently discovered, secreted lncRNAs, Gadlor1 and Gadlor2 ( Gadlor 1/2 ). Methods We …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2022 article OpenAlex

Skeletal muscle derived Musclin protects the heart during pathological overload

Malgorzata Szaroszyk, Badder Kattih, Abel Martín-Garrido, Felix A. Trogisch et autres

Cachexia is associated with poor prognosis in chronic heart failure patients, but the underlying mechanisms of cachexia triggered disease progression remain poorly understood. Here, we investigate whether the dysregulation of myokine expression from wasting skeletal muscle exaggerates heart failure. RNA sequencing from …

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82 citations Nature Communications
Accès ouvert 2018 article OpenAlex

Monophosphorylation of cardiac troponin-I at Ser-23/24 is sufficient to regulate cardiac myofibrillar Ca2+ sensitivity and calpain-induced proteolysis

Abel Martín-Garrido, Brandon J. Biesiadecki, Hussam E. Salhi, Yasin Shaifta et autres

The acceleration of myocardial relaxation produced by β-adrenoreceptor stimulation is mediated in part by protein kinase A (PKA)-mediated phosphorylation of cardiac troponin-I (cTnI), which decreases myofibrillar Ca 2+ sensitivity. Previous evidence suggests that phosphorylation of both Ser-23 and Ser-24 in cTnI is …

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39 citations Journal of Biological Chemistry
Accès ouvert 2018 conference-abstract OpenAlex

P457Monophosphorylation of cardiac troponin-I at Ser23/24 regulates cardiac myofibrillar Ca2+ sensitivity and modulates calpain-induced proteolysis

Jonathan C. Kentish, Abel Martín-Garrido, Brandon J. Biesiadecki, Hussam E. Salhi et autres

Funding Acknowledgements: Supported by grants from the British Heart Foundation (to JK and MA) and NIH (to BJB and YG) Introduction: The positive inotropic and lusitropic effects of β-adrenoceptor stimulation are mediated in part by protein kinase A (PKA)-mediated phosphorylation of cardiac …

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0 citations Cardiovascular Research

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