Aller au contenu principal
Profil bibliographique

Mendy Black

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

13Publications signalées
542Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Neurotransmitter Receptor Influence on BehaviorNeuroscience and Neuropharmacology ResearchPeroxisome Proliferator-Activated ReceptorsAlcohol Consumption and Health EffectsNeuroinflammation and Neurodegeneration Mechanisms

Les publications récentes

Accès ouvert 2019 article OpenAlex

Scn4b regulates the hypnotic effects of ethanol and other sedative drugs

Yuri A. Blednov, Michal Bajo, Amanda J. Roberts, Adriana J. Da Costa et autres

The voltage-gated sodium channel subunit β4 (SCN4B) regulates neuronal activity by modulating channel gating and has been implicated in ethanol consumption in rodent models and human alcoholics. However, the functional role for Scn4b in ethanol-mediated behaviors is unknown. We determined if genetic …

us (code pays fourni par la source)

8 citations Genes Brain & Behavior
Accès ouvert 2017 article OpenAlex

Sedative and Motor Incoordination Effects of Ethanol in Mice Lacking CD14, TLR2, TLR4, or MyD88

Yuri A. Blednov, Mendy Black, Jillian M. Benavidez, Adriana Da Costa et autres

BACKGROUND: In our companion article, we examined the role of MyD88-dependent signaling in ethanol (EtOH) consumption in mice lacking key components of this inflammatory pathway and observed differential effects on drinking. Here, we studied the role of these same signaling components in …

us (code pays fourni par la source)

31 citations Alcoholism Clinical and Experimental Research
Accès ouvert 2017 article OpenAlex

Ethanol Consumption in Mice Lacking CD14, TLR2, TLR4, or MyD88

Yuri A. Blednov, Mendy Black, Julia Chernis, Adriana Da Costa et autres

BACKGROUND: Molecular and behavioral studies support a role for innate immune proinflammatory pathways in mediating the effects of alcohol. Increased levels of Toll-like receptors (TLRs) have been observed in animal models of alcohol consumption and in human alcoholics, and many of these …

us (code pays fourni par la source)

67 citations Alcoholism Clinical and Experimental Research
Accès ouvert 2016 article OpenAlex

Inhibition of IKKβ Reduces Ethanol Consumption in C57BL/6J Mice

Jay Truitt, Yuri A. Blednov, Jillian M. Benavidez, Mendy Black et autres

Abstract Proinflammatory pathways in neuronal and non-neuronal cells are implicated in the acute and chronic effects of alcohol exposure in animal models and humans. The nuclear factor-κB (NF-κB) family of DNA transcription factors plays important roles in inflammatory diseases. The kinase IKKβ …

us (code pays fourni par la source)

40 citations eNeuro
Accès ouvert 2016 article OpenAlex

PPAR Agonists: I. Role of Receptor Subunits in Alcohol Consumption in Male and Female Mice

Yuri A. Blednov, Mendy Black, Jillian M. Benavidez, Eleni E. Stamatakis et autres

BACKGROUND: Several peroxisome proliferator-activated receptor (PPAR) agonists reduce voluntary alcohol consumption in rodent models, and evidence suggests that PPARα and γ subunits play an important role in this effect. To define the subunit dependence of this action, we tested selective PPARα and …

us (code pays fourni par la source)

31 citations Alcoholism Clinical and Experimental Research
Accès ouvert 2016 article OpenAlex

PPAR Agonists: II. Fenofibrate and Tesaglitazar Alter Behaviors Related to Voluntary Alcohol Consumption

Yuri A. Blednov, Mendy Black, Jillian M. Benavidez, Eleni E. Stamatakis et autres

BACKGROUND: In the accompanying article, we showed that activation of peroxisome proliferator-activated receptor alpha (PPARα) signaling by fenofibrate and tesaglitazar decreases ethanol (EtOH) consumption in mice. In this study, we determined the role of these PPAR agonists in EtOH-related behaviors and other …

us (code pays fourni par la source)

33 citations Alcoholism Clinical and Experimental Research
Accès ouvert 2014 article OpenAlex

Peroxisome Proliferator‐Activated Receptorsαandγare Linked with Alcohol Consumption in Mice and Withdrawal and Dependence in Humans

Yuri A. Blednov, Jillian M. Benavidez, Mendy Black, Laura B. Ferguson et autres

BACKGROUND: Peroxisome proliferator-activated receptor (PPAR) agonists reduce voluntary ethanol (EtOH) consumption in rat models and are promising therapeutics in the treatment for drug addictions. We studied the effects of different classes of PPAR agonists on chronic EtOH intake and preference in mice …

us (code pays fourni par la source)

96 citations Alcoholism Clinical and Experimental Research
Accès ouvert 2014 article OpenAlex

Inhibition of phosphodiesterase 4 reduces ethanol intake and preference in C57BL/6J mice

Yuri A. Blednov, Jillian M. Benavidez, Mendy Black, Robert Adron Harris

Some anti-inflammatory medications reduce alcohol consumption in rodent models. Inhibition of phosphodiesterases (PDE) increases cAMP and reduces inflammatory signaling. Rolipram, an inhibitor of PDE4, markedly reduced ethanol intake and preference in mice and reduced ethanol seeking and consumption in alcohol-preferring fawn-hooded rats …

us (code pays fourni par la source)

76 citations Frontiers in Neuroscience
Accès ouvert 2014 article OpenAlex

GABAA Receptors Containing ρ1 Subunits Contribute to In Vivo Effects of Ethanol in Mice

Yuri A. Blednov, Jillian M. Benavidez, Mendy Black, Courtney R. Leiter et autres

GABAA receptors consisting of ρ1, ρ2, or ρ3 subunits in homo- or hetero-pentamers have been studied mainly in retina but are detected in many brain regions. Receptors formed from ρ1 are inhibited by low ethanol concentrations, and family-based association analyses have linked …

us, au (code pays fourni par la source)

58 citations PLoS ONE

BNTIC News n’est pas le producteur de ces données. Les publications sont interrogées à la demande dans Crossref, OpenAIRE, DOAJ, Europe PMC, HAL, DataCite, AfricArXiv, ROR et la Banque mondiale, sans clé d’accès. OpenAlex reste optionnel. Aucun service payant n’est nécessaire et aucune donnée externe n’est enregistrée en base. Consulter les sources et leurs limites.