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Profil bibliographique

Elham Hasheminasabgorji

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

5Publications signalées
26Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Multiple Myeloma Research and TreatmentsGenetics and Neurodevelopmental DisordersPolyamine Metabolism and ApplicationsGenomics and Chromatin DynamicsEpigenetics and DNA Methylation

Les publications récentes

Accès ouvert 2025 article OpenAlex

EZHIP boosts neuronal-like synaptic gene programs and depresses polyamine metabolism

Elham Hasheminasabgorji, Huey‐Miin Chen, Taylor A. Gatesman, Subhi Talal Younes et autres

It is currently understood that the characteristic loss of the repressive histone mark H3K27me3 in PFA ependymoma and diffuse midline glioma (DMG) are caused by complementary mechanisms mediated by EZHIP and the oncohistone H3K27M, respectively. To support the complementarity of these mechanisms, …

ca, us (code pays fourni par la source)

2 citations Acta Neuropathologica Communications
Accès ouvert 2025 preprint OpenAlex

EZHIP boosts neuronal-like synaptic gene programs and depresses polyamine metabolism

Elham Hasheminasabgorji, Huey‐Miin Chen, Taylor A. Gatesman, Subhi Talal Younes et autres

ABSTRACT It is currently understood that the characteristic loss of the repressive histone mark H3K27me3 in PFA ependymoma and diffuse midline glioma (DMG) are caused by complementary mechanisms mediated by EZHIP and the oncohistone H3K27M, respectively. To support the complementarity of these …

ca, us (code pays fourni par la source)

0 citations bioRxiv (Cold Spring Harbor Laboratory)
2025 conference-abstract OpenAlex

Abstract 902: Examining the impact of chromosome 1q-located gamma secretase subunits in the resistance to BCMA-targeted therapies in multiple myeloma patients

Sejal Chikhale, Mansour Poorebrahim, Holly Lee, Elham Hasheminasabgorji et autres

Abstract Multiple Myeloma (MM) is an incurable hematological malignancy originating from abnormal plasma cells in the bone marrow. A common genetic abnormality in MM is the gain or amplification of chromosome arm 1q (chr1q), present in 30-40% of MM patients and typically …

ca (code pays fourni par la source)

0 citations Cancer Research
Accès ouvert 2024 article OpenAlex

TULIPs decorate the three-dimensional genome of PFA ependymoma

Michael J.G. Johnston, John J. Y. Lee, Bo Hu, Ana Antic Nikolic et autres

Posterior fossa group A (PFA) ependymoma is a lethal brain cancer diagnosed in infants and young children. The lack of driver events in the PFA linear genome led us to search its 3D genome for characteristic features. Here, we reconstructed 3D genomes …

ca, kr, us, jp, hu, pl (code pays fourni par la source)

24 citations Cell

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