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Profil bibliographique

Katariina Granath

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

3Publications signalées
22Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Genetic Neurodegenerative DiseasesFetal and Pediatric Neurological DisordersMitochondrial Function and PathologyNeurological disorders and treatmentsUbiquitin and proteasome pathways

Les publications récentes

Accès ouvert 2025 article OpenAlex

A Novel Homozygous KIF1C Variant in 2 Cases of Spastic Ataxia Type 2

Katariina Granath, Salla M. Kangas, Sanna Huhtaniska, Maria Helena Suo-Palosaari et autres

Objectives: gene. Methods: Two unrelated individuals with early-onset spastic ataxia were evaluated for genetic etiology by exome sequencing. Case reports were compiled through a medical chart review. Two cellular models were established to assess variant pathogenicity. Results: variant included retinal dysfunction detected …

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1 citation Neurology Genetics
Accès ouvert 2025 article OpenAlex

Phenotypic Heterogeneity in Genetic and Acquired Pediatric Cerebellar Disorders

Katariina Granath, Sanna Huhtaniska, Juulia Ellonen, Tytti M.-L. Pokka et autres

BACKGROUND: The genetic landscape of pediatric cerebellar disorders (PCDs) in Finland is undefined. OBJECTIVES: The objective was to define epidemiological, clinical, neuroradiological, and genetic characteristics of PCDs in Northern Finland. METHODS: A longitudinal population-based cohort study of children with a movement disorder …

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3 citations Movement Disorders
Accès ouvert 2023 article OpenAlex

Detailed Analysis of ITPR1 Missense Variants Guides Diagnostics and Therapeutic Design

Jussi‐Pekka Tolonen, Ricardo Parolin Schnekenberg, Simon J. McGowan, David Sims et autres

BACKGROUND: R1), a critical player in cerebellar intracellular calcium signaling. Pathogenic missense variants in ITPR1 cause congenital spinocerebellar ataxia type 29 (SCA29), Gillespie syndrome (GLSP), and severe pontine/cerebellar hypoplasia. The pathophysiological basis of the different phenotypes is poorly understood. OBJECTIVES: We aimed …

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18 citations Movement Disorders

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