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Profil bibliographique

Eric Mi

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

7Publications signalées
61Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Clostridium difficile and Clostridium perfringens researchGut microbiota and healthUbiquitin and proteasome pathwaysChemokine receptors and signalingCardiovascular Effects of Exercise

Les publications récentes

Accès ouvert 2023 article OpenAlex

Myofibroblast senescence promotes arrhythmogenic remodeling in the aged infarcted rabbit heart

Brett Baggett, Kevin R. Murphy, Elif Sengun, Eric Mi et autres

Progressive tissue remodeling after myocardial infarction (MI) promotes cardiac arrhythmias. This process is well studied in young animals, but little is known about pro-arrhythmic changes in aged animals. Senescent cells accumulate with age and accelerate age-associated diseases. Senescent cells interfere with cardiac …

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12 citations eLife
Accès ouvert 2022 article OpenAlex

Characterization of NanR Regulation of Sialidase Production, Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strains Carrying a Chromosomal Enterotoxin Gene

Jihong Li, Eric Mi, Arhat Pradhan, Bruce A. McClane

Clostridium perfringens type F food poisoning (FP) strains produce C. perfringens enterotoxin (CPE) to cause a common bacterial food-borne illness in the United States. During FP, CPE is synthesized in the intestines when C. perfringens sporulates. Besides CPE, FP strains also produce …

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3 citations Toxins
Accès ouvert 2021 preprint OpenAlex

Myofibroblast Senescence Promotes Arrhythmogenic Remodeling in the Aged Infarcted Rabbit Heart

Brett Baggett, Kevin R. Murphy, Elif Sengun, Eric Mi et autres

Abstract Progressive tissue remodeling after myocardial infarction (MI) promotes cardiac arrhythmias. This process is well studied in young animals, but little is known about pro-arrhythmic changes in aged animals. Senescent cells accumulate with age and accelerate age-associated diseases. Senescent cells interfere with …

us, gb (code pays fourni par la source)

0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2019 article OpenAlex

Identification of an Important Orphan Histidine Kinase for the Initiation of Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strain SM101

John C. Freedman, Jihong Li, Eric Mi, Bruce A. McClane

Clostridium perfringens type F enteric diseases, which include a very common form of food poisoning and many cases of antibiotic-associated diarrhea, develop when type F strains sporulate and produce C. perfringens enterotoxin (CPE) in the intestines. Spores are also important for transmission …

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22 citations mBio
Accès ouvert 2018 article OpenAlex

Extracellular Ubiquitin(1–76) and Ubiquitin(1–74) Regulate Cardiac Fibroblast Proliferation

Edwin K. Jackson, Eric Mi, Vladimir B. Ritov, Delbert G. Gillespie

SDF-1α (stromal cell–derived factor-1α) is a CXCR4-receptor agonist and DPP4 (dipeptidyl peptidase 4) substrate. SDF-1α, particularly when combined with sitagliptin to block the metabolism of SDF-1α by DPP4, stimulates proliferation of cardiac fibroblasts via the CXCR4 receptor; this effect is greater in …

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9 citations Hypertension
Accès ouvert 2018 article OpenAlex

NanR Regulates Sporulation and Enterotoxin Production by Clostridium perfringens Type F Strain F4969

Eric Mi, Jihong Li, Bruce A. McClane

Clostridium perfringens type F strains, which produce C. perfringens enterotoxin (CPE), are a major cause of gastrointestinal infections, including the second most prevalent bacterial foodborne illness and 5 to 10% cases of antibiotic-associated diarrhea. Virulence of type F strains is primarily ascribable …

us (code pays fourni par la source)

15 citations Infection and Immunity
2015 conference-abstract OpenAlex

Abstract P197: Extracellular Ubiquitin Blocks CXCL12-Induced Proliferation of Cardiac Fibroblasts

Edwin K. Jackson, Eric Mi, Vladimir B. Ritov, Delbert G. Gillespie

CXCR4 receptors mediate in part hypertension-induced cardiac fibrosis. This suggests that endogenous CXCR4-receptor agonists stimulate cardiac fibroblast (CF) proliferation; however, this possibility is unexplored. Although CXCL12 (aka SDF-1α) is the best described endogenous CXCR4 agonist, ubiquitin 1-76 (U-76) exists in the extracellular …

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0 citations Hypertension

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