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Profil bibliographique

Elizabeth J. Allenger

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

9Publications signalées
441Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Neuroinflammation and Neurodegeneration MechanismsImmune cells in cancerAlzheimer's disease research and treatmentsSingle-cell and spatial transcriptomicsAtherosclerosis and Cardiovascular Diseases

Les publications récentes

Accès ouvert 2023 article OpenAlex

APOE genotype modifies microglial immunometabolism

Nicholas A. Devanney, Sangderk Lee, Holden C. Williams, Elizabeth J. Allenger et autres

Abstract Background Metabolic dysfunction and neuroinflammation characterize Alzheimer’s disease (AD), but it is unclear if these two facets of the disease are linked. The E4 allele of Apolipoprotein E (APOE) is the strongest genetic risk factor for late‐onset AD and is associated …

us (code pays fourni par la source)

0 citations Alzheimer s & Dementia
Accès ouvert 2023 article OpenAlex

APOE modulates microglial immunometabolism in response to age, amyloid pathology, and inflammatory challenge

Sangderk Lee, Nicholas A. Devanney, Lesley R. Golden, Cathryn T Smith et autres

The E4 allele of Apolipoprotein E (APOE) is associated with both metabolic dysfunction and a heightened pro-inflammatory response: two findings that may be intrinsically linked through the concept of immunometabolism. Here, we combined bulk, single-cell, and spatial transcriptomics with cell-specific and spatially …

us (code pays fourni par la source)

210 citations Cell Reports
2022 article OpenAlex

APOE genotype alters the lipid droplet proteome and modulates droplet dynamics

Cathryn T Smith, Cassi M. Friday, Nicholas A. Devanney, Elizabeth J. Allenger et autres

Abstract Background The microglial immune response is a significant contributor to Alzheimer’s disease (AD) pathophysiology and neurodegeneration. Aging microglia accumulate lipid droplets (LDs), have high levels of reactive oxygen species, secrete pro‐inflammatory cytokines, and are defective in phagocytosis. The E4 allele of …

us (code pays fourni par la source)

0 citations Alzheimer s & Dementia
Accès ouvert 2022 article OpenAlex

APOE genotype modifies microglial immunometabolism

Nicholas A. Devanney, Sangderk Lee, Holden C. Williams, Elizabeth J. Allenger et autres

Abstract Background Metabolic dysfunction and neuroinflammation characterize Alzheimer’s disease (AD), but it is unclear if these two facets of the disease are linked. The E4 allele of Apolipoprotein E (APOE) is the strongest genetic risk factor for late‐onset AD and is associated …

us (code pays fourni par la source)

0 citations Alzheimer s & Dementia
Accès ouvert 2022 preprint OpenAlex

APOE modulates microglial immunometabolism in response to age, amyloid pathology, and inflammatory challenge

Sangderk Lee, Nicholas A. Devanney, Lesley R. Golden, Cathryn T Smith et autres

Summary The E4 allele of Apolipoprotein E ( APOE ) is associated with both metabolic dysfunction and a heightened pro-inflammatory response – two findings that may be intrinsically linked through the concept of immunometabolism. Here, we combined bulk, single-cell, and spatial transcriptomics …

us (code pays fourni par la source)

5 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2021 article OpenAlex

APOΕ4 lowers energy expenditure in females and impairs glucose oxidation by increasing flux through aerobic glycolysis

Brandon C. Farmer, Holden C. Williams, Nicholas A. Devanney, Margaret A. Piron et autres

BACKGROUND: Cerebral glucose hypometabolism is consistently observed in individuals with Alzheimer's disease (AD), as well as in young cognitively normal carriers of the Ε4 allele of Apolipoprotein E (APOE), the strongest genetic predictor of late-onset AD. While this clinical feature has been …

us (code pays fourni par la source)

92 citations Molecular Neurodegeneration
Accès ouvert 2020 preprint OpenAlex

APOE4 Lowers Energy Expenditure and Impairs Glucose Oxidation by Increasing Flux through Aerobic Glycolysis

Brandon C. Farmer, Holden C. Williams, Nicholas A. Devanney, Margaret A. Piron et autres

Abstract Cerebral glucose hypometabolism is consistently observed in individuals with Alzheimer’s disease (AD), as well as in young cognitively normal carriers of the E4 allele of Apolipoprotein E (APOE), the strongest genetic predictor of late-onset AD. While this clinical feature has been …

us (code pays fourni par la source)

4 citations bioRxiv (Cold Spring Harbor Laboratory)

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