Aller au contenu principal
Profil bibliographique

Keyu Wang

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

16Publications signalées
110Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

melanin and skin pigmentationAtherosclerosis and Cardiovascular DiseasesT-cell and B-cell ImmunologyMicroRNA in disease regulationAntifungal resistance and susceptibility

Les publications récentes

Accès ouvert 2025 article OpenAlex

Single-cell analysis of ecto-mesenchymal cells derived from the neural crest reveals the regulatory role of P75NTR in odontogenic and osteogenic differentiation

Feng Li, Yanyan Zhang, Keyu Wang, Xin Nie

BACKGROUND: The aim to this study was to explore the effect of p75NTR on the mineralization and development of maxillofacial processes. Moreover, we tried to elaborate the potential mechanism of p75NTR's effect on the odontogenic or osteogenic differentiation ability of neural crest …

cn (code pays fourni par la source)

0 citations BMC Oral Health
Accès ouvert 2019 article OpenAlex

MicroRNA‑155 inhibits the proliferation of CD8+ T�cells via upregulating regulatory T cells in vitiligo

Mingfen Lv, Zhengjun Li, Jingjing Liu, Fan Lin et autres

It has been reported that loss and degradation of epidermal melanocytes is closely associated with the pathogenesis of vitiligo. In addition, CD8+ T and regulatory T (Treg) cells serve an important role during these two processes. MicroRNA‑155 (miR‑155) is known to contribute …

cn (code pays fourni par la source)

22 citations Molecular Medicine Reports
Accès ouvert 2019 article OpenAlex

MicroRNA-664 suppresses the growth of cervical cancer cells via targeting c-Kit

Mingfen Lv, Rongying Ou, Qianwen Zhang, Fan Lin et autres

BACKGROUND: Cervical cancer is the second most common malignant cancer in women worldwide. Evidence indicated that miR-664 was significantly downregulated in cervical cancer. However, the mechanisms by which miR-664 regulates the tumorigenesis of cervical cancer remain unclear. Thus, this study aimed to …

cn (code pays fourni par la source)

13 citations Drug Design Development and Therapy
2018 article OpenAlex

Geniposide protects melanocytes from oxidative damage through the PI3K-Akt pathway

Wenjing Lü, Yu Zhao, Keyu Wang

Objective To evaluate the inhibitory effect of geniposide on the oxidative damage to in vitro cultured human melanocytes, and to explore the role of PI3K-Akt signaling pathway in this inhibitory effect. Methods Epidermal melanocytes were isolated from the circumcised foreskin of healthy …

cn (code pays fourni par la source)

0 citations Chinese Journal of Dermatology
Accès ouvert 2018 article OpenAlex

Protective effects of glutamine on human melanocyte oxidative stress model

Keyu Wang, Liya Jiang, Zhen Guo, Yulong Kong et autres

BACKGROUND: Vitiligo is a disorder caused by the loss of the melanocyte activity on melanin pigment generation. Studies show that oxidative-stress induced apoptosis in melanocytes is closely related to the pathogenesis of vitiligo. Glutamine is a well known antioxidant with anti-apoptotic effects, …

cn (code pays fourni par la source)

10 citations Indian Journal of Dermatology Venereology and Leprology
2014 article OpenAlex

Differential expression analysis of mi RNA in peripheral blood mononuclear cells of patients with non‐segmental vitiligo

Yi Wang, Keyu Wang, Jianhua Liang, Hong Yang et autres

Vitiligo is a common depigmentary skin disease that may follow a pattern of multifactorial inheritance. The essential factors of its immunopathogenesis is thought to be the selective destruction of melanocytes. As a new class of microregulators of gene expression, miRNA have been …

cn (code pays fourni par la source)

44 citations The Journal of Dermatology

BNTIC News n’est pas le producteur de ces données. Les publications sont interrogées à la demande dans Crossref, OpenAIRE, DOAJ, Europe PMC, HAL, DataCite, AfricArXiv, ROR et la Banque mondiale, sans clé d’accès. OpenAlex reste optionnel. Aucun service payant n’est nécessaire et aucune donnée externe n’est enregistrée en base. Consulter les sources et leurs limites.