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Profil bibliographique

Lucia Juana‐López

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

6Publications signalées
316Citations signalées
0Affiliations récentes

Les domaines associés

Glycogen Storage Diseases and MyoclonusGenetics and Neurodevelopmental DisordersNeurological disorders and treatmentsLysosomal Storage Disorders ResearchHemoglobinopathies and Related Disorders

Les publications récentes

Accès ouvert 2025 article OpenAlex

Functional and structural characterization of mouse Factor H-related B protein unveils a novel dimerization domain shared by FHR-B and FH

Bárbara Márquez Tirado, Lucia Juana‐López, Elena Goicoechea de Jorge, Santiago Rodrı́guez de Córdoba et autres

Factor H-related proteins (FHRs) are found in mice, but their equivalence to human FHRs remains uncertain. This study identifies three FHRs in mouse plasma (FHR-B, FHR-C, and FHR-E) and focuses on characterizing FHR-B. Using purified plasma proteins and recombinant mutants, FHR-B was …

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1 citation Frontiers in Immunology
Accès ouvert 2017 article OpenAlex

Abnormal glycogen chain length pattern, not hyperphosphorylation, is critical in Lafora disease

Felix Nitschké, Mitchell Anthony Sullivan, Peixiang Wang, Xiaochu Zhao et autres

Lafora disease (LD) is a fatal progressive epilepsy essentially caused by loss-of-function mutations in the glycogen phosphatase laforin or the ubiquitin E3 ligase malin. Glycogen in LD is hyperphosphorylated and poorly hydrosoluble. It precipitates and accumulates into neurotoxic Lafora bodies (LBs). The …

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93 citations EMBO Molecular Medicine
Accès ouvert 2014 article OpenAlex

The phosphatase activity of laforin is dispensable to rescue Epm2a−/− mice from Lafora disease

Javier Machín Gayarre, Lara Durán-Trío, Olga Criado‐García, Carmen Aguado et autres

Lafora progressive myoclonus epilepsy (Lafora disease) is a fatal autosomal recessive neurodegenerative disorder characterized by the presence of glycogen-like intracellular inclusions called Lafora bodies. The vast majority of patients carry mutations in either the EPM2A or EPM2B genes, encoding laforin, a glucan …

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45 citations Brain
Accès ouvert 2012 erratum OpenAlex

Erratum to

Erwin Knecht, Olga Criado‐García, Carmen Aguado, Javier Machín Gayarre et autres

In the Acknowledgments of our report we unintentionally omitted to include a sentence stating that J.G. holds a postdoctoral fellowship (JAE-Doc) from the Program “Junta para la Ampliación de Estud...

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1 citation Autophagy
Accès ouvert 2012 article OpenAlex

Malin knockout mice support a primary role of autophagy in the pathogenesis of Lafora disease

Erwin Knecht, Olga Criado‐García, Carmen Aguado, Javier Machín Gayarre et autres

Lafora disease (LD), a fatal neurodegenerative disorder characterized by intracellular inclusions called Lafora bodies (LBs), is caused by recessive loss-of-function mutations in the genes encoding either laforin or malin. Previous studies suggested a role of these proteins in regulating glycogen biosynthesis, in …

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22 citations Autophagy
Accès ouvert 2011 article OpenAlex

Lafora bodies and neurological defects in malin-deficient mice correlate with impaired autophagy

Olga Criado‐García, Carmen Aguado, Javier Machín Gayarre, Lara Durán-Trío et autres

Lafora disease (LD), a fatal neurodegenerative disorder characterized by the presence of intracellular inclusions called Lafora bodies (LBs), is caused by loss-of-function mutations in laforin or malin. Previous studies suggested a role of these proteins in the regulation of glycogen biosynthesis, in …

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154 citations Human Molecular Genetics

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