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Profil bibliographique

Belgin Yalçın

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

45Publications signalées
1896Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Neurogenesis and neuroplasticity mechanismsNeuroscience and Neuropharmacology ResearchGlioma Diagnosis and TreatmentNeuroinflammation and Neurodegeneration MechanismsCancer, Stress, Anesthesia, and Immune Response

Les publications récentes

Accès ouvert 2026 article OpenAlex

Gliomas phenocopy an inborn error of metabolism to drive neuronal activity and tumor growth

Kalil G. Abdullah, Kenji Miki, Charles K. Edgar, Shuangcheng Alivia Wu et autres

receptors and causing depolarizing currents in glioma-associated neurons with dysregulated chloride homeostasis. Depleting tumoral GAA decreased electrochemical activity, neuron-glioma interactions, and tumor aggressiveness. Our findings unveil a mechanism linking cancer metabolism with cancer neuroscience and leverage human genetics to nominate GAA synthesis …

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0 citations Cell
Accès ouvert 2026 preprint OpenAlex

Juvenile influenza can impair myelin development and adult behavior through chemokine signaling in mice

Karen Malacon, Kiarash Shamardani, Sophie Artandi, Lijun Ni et autres

SUMMARY Brain development, especially developmental myelination, continues through young adulthood. Concordantly, children may be particularly vulnerable to neural-immune challenges. To investigate the consequences of major childhood immune challenges, juvenile mice were exposed to respiratory influenza (H1N1) infection. White matter-specific microglial reactivity accompanied …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2026 article OpenAlex

Neuroligin-3–CSPG4 interaction maintains oligodendrocyte precursor cell progenitor state and promotes glioma proliferation through mechanotransduction

Yoon Seok Kim, Shawn Gillespie, Anna C. Geraghty, Belgin Yalçın et autres

Glioma pathophysiology is robustly regulated by interactions with neurons. Key to these interactions is the role of neuroligin-3 (NLGN3), a synaptic adhesion molecule shed in response to neuronal activity that functions as a paracrine factor crucial for glioma growth. Here we elucidate …

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0 citations Nature Neuroscience
Accès ouvert 2025 preprint OpenAlex

Serotonergic neuron-glioma interactions drive high-grade glioma pathophysiology

Richard Drexler, Belgin Yalçın, Rebecca Mancusi, Abigail Rogers et autres

Abstract High-grade gliomas are lethal brain cancers that are powerfully regulated by glutamatergic neurons through activity-dependent paracrine factors and functional neuron-to-glioma synapses. Here, we report that serotonergic neurons promote the proliferation of high-grade gliomas throughout the brain. Serotonergic neuronal activity drives circuit-specific …

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3 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 article OpenAlex

CNSC-36. PSILOCYBIN INDUCES SUSTAINED GLIOMA GROWTH THROUGH SEROTONERGIC AND TRKB PATHWAYS

Richard Drexler, Belgin Yalçın, Rebecca Mancusi, Abigail Rogers et autres

Abstract High-grade gliomas are the most aggressive form of brain tumors, and neuronal activity has emerged as a driver of glioma pathophysiology. Activity-dependent glioma growth results from paracrine factor signaling and bona fide neuron-to-glioma synapses that integrate glioma cells into brain-wide neuronal …

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0 citations Neuro-Oncology
Accès ouvert 2025 conference-abstract OpenAlex

CNSC-68. LEVETIRACETAM THERAPEUTICALLY TARGETS GABAERGIC SYNAPSES IN DIFFUSE MIDLINE GLIOMA

Tara Barron, Belgin Yalçın, Minhui Su, Aaron Mochizuki et autres

Abstract Pediatric high-grade gliomas (HGGs) are the leading cause of brain cancer-related death in children. HGGs include distinct subtypes defined by anatomical location as well as molecular characteristics that stratify into H3K27M-altered diffuse midline gliomas (DMG) and hemispheric HGGs such as isocitrate …

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0 citations Neuro-Oncology
Accès ouvert 2025 article OpenAlex

Neuronal activity-dependent mechanisms of small cell lung cancer pathogenesis

Solomiia Savchuk, Kaylee M. Gentry, Wengang Wang, Elana Carleton et autres

Abstract Neural activity is increasingly recognized as a crucial regulator of cancer growth. In the brain, neuronal activity robustly influences glioma growth through paracrine mechanisms 1 and by electrochemical integration of malignant cells into neural circuitry via neuron-to-glioma synapses 2,3 . Outside …

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81 citations Nature
Accès ouvert 2025 article OpenAlex

CNSC-06. Brainstem neuromodulatory neurons promote glioma growth locally and via long-range projections to midline and cortical structures

Richard Drexler, Antonia Drinnenberg, Avishai Gavish, Belgin Yalçın et autres

Abstract Neuronal activity robustly drives the progression of diffuse midline gliomas (DMG), mediated through paracrine and synaptic neuron-to-glioma interactions. To date, research has primarily focused on glutamatergic and GABAergic neurons, while the impact of neuromodulatory neuron subpopulations and their long-range projections remain …

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0 citations Neuro-Oncology Pediatrics
Accès ouvert 2025 preprint OpenAlex

Neuroligin-3 interaction with CSPG4 regulates normal and malignant glial precursors through PIEZO1

Shawn Gillespie, Yoon Seok Kim, Anna C. Geraghty, Belgin Yalçın et autres

Abstract Glioma pathophysiology is robustly regulated by interactions with neurons. Key to these interactions is the role of neuroligin-3 (NLGN3), a synaptic adhesion molecule shed in response to neuronal activity 1–5 that functions as a paracrine factor crucial for glioma growth. Here, …

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3 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 article OpenAlex

Cholinergic neuronal activity promotes diffuse midline glioma growth through muscarinic signaling

Richard Drexler, Antonia Drinnenberg, Avishai Gavish, Belgin Yalçın et autres

Glutamatergic neuronal activity promotes proliferation of both oligodendrocyte precursor cells (OPCs) and gliomas, including diffuse midline glioma (DMG). However, the role of neuromodulatory brainstem neurons projecting to midline structures where DMGs arise remains unexplored. Here, we demonstrate that midbrain cholinergic neuronal activity …

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53 citations Cell
Accès ouvert 2025 article OpenAlex

GABAergic neuron-to-glioma synapses in diffuse midline gliomas

Tara Barron, Belgin Yalçın, Minhui Su, Youkyeong Gloria Byun et autres

Abstract High-grade gliomas (HGGs) are the leading cause of brain cancer-related death. HGGs include clinically, anatomically and molecularly distinct subtypes that stratify into diffuse midline gliomas (DMGs), such as H3K27M -altered diffuse intrinsic pontine glioma, and hemispheric HGGs, such as IDH wild-type …

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135 citations Nature
Accès ouvert 2024 conference-abstract OpenAlex

CNSC-61. NEURONAL-ACTIVITY DEPENDENT MECHANISMS OF SMALL CELL LUNG CANCER PATHOGENESIS

Solomiia Savchuk, Wengang Wang, Kaylee M. Gentry, Elana Carleton et autres

Abstract Neural activity is increasingly recognized as a crucial regulator of cancer growth. In the brain, neuronal activity robustly influences glioma growth both through paracrine mechanisms and through electrochemical integration of malignant cells into neural circuitry via neuron-to-glioma synapses. Outside of the …

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0 citations Neuro-Oncology

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