VDAC1 Upregulation Induces Hyperexcitability of Nociceptive Sensory Neurons Via Enhanced Mitochondrial Atp Efflux in Neuropathic Pain
Fengrun Sun, Ning Yu, Liang Yu, Xiaofen Tian et autres
Neuropathic pain results from aberrant sensory processing following nerve injury. Voltage-dependent anion channels (VDACs), a family of ATP gatekeepers in the outer mitochondrial membrane, are central to cellular metabolism and action potential generation, yet their roles in peripheral neuropathic pain remain poorly …
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Advanced Science