CIRKIL Assembles the PKM2 Homotetramer to Exacerbate Myocardial Fibrosis after Myocardial Infarction
Wei Yuan, Yizheng Wang, Bo Meng, Xiaoxuan Zhu et autres
During the healing phase after myocardial infarction (MI), various stimulatory factors activate the transformation of cardiac fibroblasts (CFs) into myofibroblasts, leading to collagen synthesis and cardiac fibrosis. Here, we investigated the role of the cardiac ischemia‒reperfusion-associated Ku70-interacting long noncoding RNA ( CIRKIL …
cn (code pays fourni par la source)