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Profil bibliographique

Selin Adakli

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

4Publications signalées
3Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Nuclear Structure and FunctionDNA Repair MechanismsMicrotubule and mitosis dynamicsRNA Research and SplicingCellular transport and secretion

Les publications récentes

Accès ouvert 2025 article OpenAlex

Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels

Veronica Cherdyntseva, Daniel González‐Acosta, Patricia Ubieto‐Capella, M. do C. Rodrigues et autres

Mild replication interference is a consolidated strategy for cancer chemotherapy. Tolerance to mild replication stress (RS) relies on active fork slowing, mediated by transient fork reversal and RECQ1-assisted restart, and modulated by PARP1 and nuclear architectural components via yet-elusive mechanisms. We combined …

ch, nl, ca (code pays fourni par la source)

1 citation Nature Communications
Accès ouvert 2025 article OpenAlex

Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels

Veronica Cherdyntseva, Joanna M. Paulson, Daniel González-Acosta, Patricia Ubieto‐Capella et autres

Mild replication interference is a consolidated strategy for cancer chemotherapy. Tolerance to mild replication stress (RS) relies on active fork slowing, mediated by transient fork reversal and RECQ1-assisted restart, and modulated by PARP1 and nuclear architectural components via yet-elusive mechanisms. We combined …

0 citations Zurich Open Repository and Archive (University of Zurich)
Accès ouvert 2025 article OpenAlex

Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels

Veronica Cherdyntseva, Joanna M. Paulson, Daniel González-Acosta, Patricia Ubieto‐Capella et autres

Mild replication interference is a consolidated strategy for cancer chemotherapy. Tolerance to mild replication stress (RS) relies on active fork slowing, mediated by transient fork reversal and RECQ1-assisted restart, and modulated by PARP1 and nuclear architectural components via yet-elusive mechanisms. We combined …

1 citation Zurich Open Repository and Archive (University of Zurich)
Accès ouvert 2025 preprint OpenAlex

Nucleoplasmic Lamin A/C controls replication fork restart upon stress by modulating local H3K9me3 and ADP-ribosylation levels

Veronica Cherdyntseva, Selin Adakli, Jean‐Philippe Gagné, Moses Aouami et autres

Abstract Mild replication interference is a consolidated strategy for cancer chemotherapy. Tolerance to mild replication stress (RS) relies on active fork slowing, mediated by transient fork reversal and RECQ1-assisted restart, and modulated by PARP1 and nuclear architectural components via yet-elusive mechanisms. We …

ch, nl, ca (code pays fourni par la source)

1 citation bioRxiv (Cold Spring Harbor Laboratory)

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