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Profil bibliographique

Caitlin Hall

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

6Publications signalées
50Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Cardiomyopathy and Myosin StudiesLysosomal Storage Disorders ResearchAtrial Fibrillation Management and OutcomesParkinson's Disease Mechanisms and TreatmentsCardiac electrophysiology and arrhythmias

Les publications récentes

Accès ouvert 2026 article OpenAlex

A titin truncating variant linked to atrial fibrillation increases atrial profibrotic signalling and cholinergic sensitivity

Max J. Cumberland, Albert Dasí, Naeramit Sontayananon, Alan Marcus et autres

AIMS: Titin truncating variants (TTNtv) are a major genetic cause of dilated cardiomyopathy (DCM), accounting for approximately 25% of familial cases. Atrial fibrillation (AF) frequently occurs in DCM patients carrying TTNtv and may precede overt ventricular dysfunction, suggesting an atrial-specific disease mechanism. …

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3 citations Cardiovascular Research
Accès ouvert 2025 article OpenAlex

Early Atrial Remodeling Drives Arrhythmia in Fabry Disease

Ashwin Roy, Christopher O’Shea, Albert Dasí, Leena K. Patel et autres

BACKGROUND: Fabry disease (FD) is an X-linked lysosomal storage disorder caused by α-Gal A (α-galactosidase A) deficiency, resulting in multiorgan accumulation of sphingolipid, namely globotriaosylceramide. This triggers ventricular myocardial hypertrophy, fibrosis, and inflammation, driving arrhythmia and sudden death. Atrial fibrillation is common, …

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5 citations Circulation Arrhythmia and Electrophysiology
Accès ouvert 2024 preprint OpenAlex

Early atrial remodelling drives arrhythmia in Fabry Disease

Ashwin Roy, Christopher O’Shea, Albert Dasi I Martinez, Leena K. Patel et autres

ABSTRACT Background Fabry disease (FD) is an X-linked lysosomal storage disorder caused by α-galactosidase A (α-Gal A) deficiency, resulting in multi-organ accumulation of sphingolipid, namely globotriaosylceramide (Gb3). This triggers ventricular myocardial hypertrophy, fibrosis, and inflammation, driving arrhythmia and sudden death, a common …

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1 citation bioRxiv (Cold Spring Harbor Laboratory)

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