Supplementary Data from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Supplementary Tables and Supplementary Materials and Methods
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Robin Kit‐Ho Lai
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40Publications signalées
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Les domaines associés
Cancer, Hypoxia, and MetabolismEicosanoids and Hypertension PharmacologySulfur Compounds in BiologyCancer, Lipids, and MetabolismEpigenetics and DNA Methylation
Les publications récentes
Supplementary Figure 5 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S5. Knockdown of NDUFA4L2 increases mitochondrial membrane potential and ROS production in hypoxia.
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Supplementary Figure 1 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S1: NDUFA4L2 is over-expressed in human solid cancers.
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Supplementary Figure 7 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S7. Knockdown of NDUFA4L2 suppresses tumor growth and reduces cell migratory ability.
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Supplementary Figure 4 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S4. NDUFA4 is preferentially expressed under normoxia.
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Supplementary Figure 3 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S3. HIF-1α positively regulates NDUFA4L2 in hypoxia.
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Supplementary Figure 2 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S2. Knockdown of HIF-1α or HIF-2α, and knockout of HIF-1α are confirmed by western blot analysis.
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Supplementary Figure 6 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S6. Knockdown of NDUFA4L2 induces ROS-mediated apoptosis and suppresses cell proliferation.
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Data from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Abstract Purpose: Hepatocellular carcinoma (HCC) lacks effective curative therapy. Hypoxia is commonly found in HCC. Hypoxia elicits a series of protumorigenic responses through hypoxia-inducible factor-1 (HIF1). Better understanding of the metabolic adaptations of HCC cells during hypoxia is essential to the design …
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Data from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Abstract Purpose: Hepatocellular carcinoma (HCC) lacks effective curative therapy. Hypoxia is commonly found in HCC. Hypoxia elicits a series of protumorigenic responses through hypoxia-inducible factor-1 (HIF1). Better understanding of the metabolic adaptations of HCC cells during hypoxia is essential to the design …
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Supplementary Figure 7 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S7. Knockdown of NDUFA4L2 suppresses tumor growth and reduces cell migratory ability.
0 citations
Supplementary Figure 5 from NDUFA4L2 Fine-tunes Oxidative Stress in Hepatocellular Carcinoma
Robin Kit‐Ho Lai, Mingjing Xu, David Kung‐Chun Chiu, Aki Pui‐Wah Tse et autres
Figure S5. Knockdown of NDUFA4L2 increases mitochondrial membrane potential and ROS production in hypoxia.
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