Profil bibliographique

Michał Witt

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

145Publications signalées

3262Citations signalées

2Affiliations récentes

Institutions déclarées

Institute of Human Genetics Polish Academy of Sciences

Domaines associés

Cystic Fibrosis Research AdvancesAcute Lymphoblastic Leukemia researchNeonatal Respiratory Health ResearchGenetic and Kidney Cyst DiseasesEpigenetics and DNA Methylation

Publications récentes

Accès ouvert 2026 article

KSR2 is a novel oncogene in TAL1 subtype of T-cell acute lymphoblastic leukemia with potential clinical and prognostic implications - Supplementary Methods

Monika Drobna‐Śledzińska, Natalia Maćkowska‐Maślak, Roman Jaksik, Igor Fijałkowski et autres

0 citationsZenodo (CERN European Organization for Nuclear Research)

Accès ouvert 2026 article

KSR2 is a novel oncogene in TAL1 subtype of T-cell acute lymphoblastic leukemia with potential clinical and prognostic implications - Supplementary Methods

Monika Drobna‐Śledzińska, Natalia Maćkowska‐Maślak, Roman Jaksik, Igor Fijałkowski et autres

0 citationsZenodo (CERN European Organization for Nuclear Research)

Accès ouvert 2025 article

The lack of homozygotes with a large deletion encompassing SPAG1 and POLR2K in primary ciliary dyskinesia patients suggests the lethal effect of the loss of POLR2K protein

Alicja Rabiasz, Monika Drobna‐Śledzińska, Patrycja Kaźmierczak, Michał Witt et autres

0 citationsGenes & Diseases

2024 conference-abstract

The international registry for primary ciliary dyskinesia reveals regional variation of genotypes and significant genotype-phenotype correlations

Johanna Raidt, Sarah Riepenhausen, Israel Amirav, Rodrigo Abensur Athanazio et autres

0 citations

Accès ouvert 2024 article

THE FATE OF IMPLANTED BIOBRIDGE™ COLLAGEN MATRIX – FINDINGS IN HISTOLOGY, SCANNING, AND TRANSMISSION ELECTRON MICROSCOPY

Michał Witt, Andrej Ring, Ingo Stricker, Enrico Fruth

2 citationsLymphology

Accès ouvert 2024 article

Analyses of 1236 genotyped primary ciliary dyskinesia individuals identify regional clusters of distinct DNA variants and significant genotype–phenotype correlations

Johanna Raidt, Sarah Riepenhausen, Petra Pennekamp, Heike Olbrich et autres

72 citationsEuropean Respiratory Journal

Accès ouvert 2023 article

P340: IN SEARCH FOR GENOMIC AND TRANSCRIPTOMIC PREDICTORS OF RELAPSE IN PEDIATRIC T-ALL

Małgorzata Dawidowska, Roman Jaksik, Monika Drobna‐Śledzińska, Natalia Maćkowska‐Maślak et autres

0 citationsHemaSphere

Accès ouvert 2023 article

Genetic data protection as an indispensable element of genomic medicine development

Małgorzata Madej, Paweł Karpiński, Hayane Akopyan, Michał Witt et autres

5 citationsPolskie Archiwum Medycyny Wewnętrznej

Accès ouvert 2023 preprint

Primary Ciliary Dyskinesia Genotypes Vary Markedly Across 19 Countries and Identify Significant Genotype-Phenotype Correlations

Johanna Raidt, Sarah Riepenhausen, Petra Pennekamp, Heike Olbrich et autres

0 citationsSSRN Electronic Journal

Accès ouvert 2022 article

The hero of American and Polish nations: a molecular look at Thaddeus Kosciuszko’s cause of death suggests a contribution of endocarditis caused by Cutibacterium acnes infection

Michał Witt, Miron Tokarski, Ewa Ziętkiewicz, Arleta Lebioda et autres

2 citationsMicrobial Cell Factories

Accès ouvert 2022 article

Small RNA-Seq Reveals Similar miRNA Transcriptome in Children and Young Adults with T-ALL and Indicates miR-143-3p as Novel Candidate Tumor Suppressor in This Leukemia

Małgorzata Dawidowska, Natalia Maćkowska‐Maślak, Monika Drobna‐Śledzińska, Maria Kosmalska et autres

7 citationsInternational Journal of Molecular Sciences

Accès ouvert 2022 article

Multiomics to investigate the mechanisms contributing to repression of PTPRC and SOCS2 in pediatric T‐ALL: Focus on miR‐363‐3p and promoter methylation

Monika Drobna‐Śledzińska, Natalia Maćkowska‐Maślak, Roman Jaksik, Maria Kosmalska et autres

5 citationsGenes Chromosomes and Cancer

Profil ORCID Notice OpenAlex

BNTIC News n’est pas le producteur de ces données. Métadonnées interrogées à la demande auprès de OpenAlex (CC0). Sources et limites.