Profil bibliographique

Hyung Jun Ahn

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

72Publications signalées

6146Citations signalées

1Affiliations récentes

Les institutions déclarées

Korea Institute of Science and Technology

Les domaines associés

RNA Interference and Gene DeliveryAdvanced biosensing and bioanalysis techniquesEnzyme Structure and FunctionVirus-based gene therapy researchMicroRNA in disease regulation

Les publications récentes

Accès ouvert 2025 article OpenAlex

A T7 autogene-mediated DNA vaccine platform for SARS-CoV-2: Overcoming DNA vaccine limitations with enhanced spike mRNA production

Sang Won Cho, Sang Chul Shin, Yunju Nam, Hyung Jun Ahn

kr (code pays fourni par la source)

3 citations Journal of Controlled Release

Accès ouvert 2024 preprint OpenAlex

A T7 Autogene-Based DNA Vaccine Platform for Enhanced Cytoplasmic Production of Sars-Cov-2 Spike Mrna

Hyung Jun Ahn, Sang Won Cho, Sang Chul Shin, Yunju Nam

kr (code pays fourni par la source)

0 citations SSRN Electronic Journal

Accès ouvert 2021 article OpenAlex

siRNA Nanoparticle Targeting PD-L1 Activates Tumor Immunity and Abrogates Pancreatic Cancer Growth in Humanized Preclinical Model

Hyun Jin Ryu, Seung‐Hwan Lee, Dong‐Young Kim, Myung Ji Kim et autres

kr (code pays fourni par la source)

52 citations Cells

2021 article OpenAlex

Systemic delivery of CRISPR/Cas9 to hepatic tumors for cancer treatment using altered tropism of lentiviral vector

Sung‐Jin Lee, Young-Youb Kim, Hyung Jun Ahn

kr (code pays fourni par la source)

50 citations Biomaterials

Accès ouvert 2020 article OpenAlex

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Takahiko Akematsu, Amal O. Amer, Shizuo Akira, Hyung Jun Ahn et autres

0 citations UNC Libraries

2020 other OpenAlex

Antitumor effects of retargeted AAV2 virus on ovarian cancer in vivo

Hyung Jun Ahn

0 citations

Accès ouvert 2020 article OpenAlex

Preclinical assessment of the VEGFR inhibitor axitinib as a therapeutic agent for epithelial ovarian cancer

E Sun Paik, Tae Hyun Kim, Young‐Jae Cho, Ji‐Yoon Ryu et autres

kr (code pays fourni par la source)

32 citations Scientific Reports

2020 article OpenAlex

KSP siRNA/paclitaxel-loaded PEGylated cationic liposomes for overcoming resistance to KSP inhibitors: Synergistic antitumor effects in drug-resistant ovarian cancer

Jinju Lee, Young‐Jae Cho, Jeong‐Won Lee, Hyung Jun Ahn

kr (code pays fourni par la source)

45 citations Journal of Controlled Release

2019 article OpenAlex

Cytoplasmic expression of EGFR shRNA using a modified T7 autogene-based hybrid mRNA/DNA system induces long-term EGFR silencing and prolongs antitumor effects

Sharmin Seraj, Young‐Jae Cho, Jeong‐Won Lee, Hyung Jun Ahn

kr (code pays fourni par la source)

9 citations Biochemical Pharmacology

Accès ouvert 2019 article OpenAlex

Pharmacogenomic analysis of patient-derived tumor cells in gynecologic cancers

K. Jason, Jae Ryoung Hwang, Young‐Jae Cho, Ji-Yoon Ryu et autres

kr, us (code pays fourni par la source)

28 citations Genome biology

Accès ouvert 2019 article OpenAlex

CDK7 is a reliable prognostic factor and novel therapeutic target in epithelial ovarian cancer

Jihye Kim, Young‐Jae Cho, Ji‐Yoon Ryu, Ilseon Hwang et autres

kr, us (code pays fourni par la source)

29 citations Gynecologic Oncology

2019 article OpenAlex

PLGA Nanoparticles Codelivering siRNAs against Programmed Cell Death Protein-1 and Its Ligand Gene for Suppression of Colon Tumor Growth

Seo Young Kwak, Seonmin Lee, Hee Dong Han, Suhwan Chang et autres

kr (code pays fourni par la source)

51 citations Molecular Pharmaceutics

Profil ORCID Notice OpenAlex

BNTIC News n’est pas le producteur de ces données. Les publications sont interrogées à la demande dans Crossref, OpenAIRE, DOAJ, Europe PMC, HAL, DataCite, AfricArXiv, ROR et la Banque mondiale, sans clé d’accès. OpenAlex reste optionnel. Aucun service payant n’est nécessaire et aucune donnée externe n’est enregistrée en base. Consulter les sources et leurs limites.