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Profil bibliographique

Beau Neep

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

10Publications signalées
25Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Pulmonary Hypertension Research and TreatmentsAngiogenesis and VEGF in CancerChronic Obstructive Pulmonary Disease (COPD) ResearchCoronary Interventions and DiagnosticsCongenital Heart Disease Studies

Les publications récentes

Accès ouvert 2026 article OpenAlex

BMP9 Modulates IL-33 Signaling to Mitigate EndMT in Pulmonary Arterial Hypertension

Clarissa Becher, Esmee J. Groeneveld, Rozenn Quarck, Beau Neep et autres

BACKGROUND: Pulmonary arterial hypertension (PAH) is a progressive disorder involving disrupted BMP (bone morphogenetic protein) signaling, pulmonary inflammation, and endothelial-to-mesenchymal transition (EndMT). We hypothesized that IL (interleukin)-33 signaling contributes to PAH progression by inducing EndMT and interacting with BMP9, a key modulator …

nl, be, de, fr, us, es (code pays fourni par la source)

4 citations Hypertension
2025 conference-abstract OpenAlex

Unveiling the impact of VEGFR2/KDR modulation on pulmonary microvascular endothelial cells in COPD/emphysema

Wenjun He, Xiaoke Pan, Xiao‐Qing Sun, Beau Neep et autres

Background: Chronic Obstructive Pulmonary Disease (COPD) poses a global vascular challenge in emphysema. As VEGFR2/KDR is a shear stress sensor and pivotal for maintaining vascular integrity, we examined its role in sustaining pulmonary microvascular endothelial barrier integrity. Methods: VEGFR2/KDR expression was assessed …

nl (code pays fourni par la source)

0 citations
2025 conference-abstract OpenAlex

The role of SOX17 in the shear stress response of the arterial endothelium

Beau Neep, Laura De Heus, Xiaoke Pan, Robert Szulcek et autres

Background: Rare and common genetic variants in SOX17 worsen PAH and accelerate disease progression. Although SOX17 is known as an embryonic transcription factor for arterial differentiation and angiogenesis, its function in the adult vasculature is unclear. We hypothesized that SOX17 drives resilience …

nl, de, gb (code pays fourni par la source)

0 citations
2025 conference-abstract OpenAlex

Unveiling the Impact of VEGFR2/KDR Modulation on Pulmonary Microvascular Endothelial Cells in COPD/Emphysema

He Wen, Xiao‐Qing Sun, Beau Neep, H.J. Bogaard et autres

Abstract Background: Chronic Obstructive Pulmonary Disease (COPD) is a growing global health concern with incompletely understood pathophysiology. Loss of pulmonary microvasculature significantly contributes to emphysema development. Given VEGFR2/KDR's role in vascular homeostasis, we hypothesize that VEGFR2 is essential for maintaining lung microvascular …

nl (code pays fourni par la source)

1 citation American Journal of Respiratory and Critical Care Medicine
Accès ouvert 2025 conference-abstract OpenAlex

The Role of SOX17 in the Shear Stress Response of the Arterial Endothelium

Beau Neep, Laura E. de Heus, Xiaoke Pan, Robert Szulcek et autres

Abstract Background: Pulmonary arterial hypertension (PAH) is a fatal lung disease characterized by pulmonary vascular remodeling. Recently, genetic variations in SOX17 were shown to worsen the PAH phenotype and accelerate disease progression. Although SOX17 is a well-known embryonic endothelial-specific transcription factor that …

nl, de (code pays fourni par la source)

0 citations American Journal of Respiratory and Critical Care Medicine
Accès ouvert 2025 preprint OpenAlex

Empagliflozin Improves Mitochondrial Biogenesis and Ameliorates Experimental Pulmonary Vascular Remodeling, But May Not Benefit Patients with Pulmonary Arterial Hypertension

Keimei Yoshida, Eszter Margit Tóth, Erik Duijvelaar, Beau Neep et autres

Abstract Background The sodium glucose cotransporter 2 (SGLT2) inhibitor may improve mitochondrial biogenesis and attenuate pulmonary vascular remodeling in pulmonary arterial hypertension (PAH). We investigated the impact of empagliflozin in PAH. Methods Lung sections and primary cell cultures isolated from microvascular endothelial …

jp, nl, us (code pays fourni par la source)

1 citation medRxiv
Accès ouvert 2025 article OpenAlex

The lncRNA DSCR9 is modulated in pulmonary arterial hypertension endothelial cell models and is associated with alterations in the nitric oxide pathway

Nadia Bernardi, Beau Neep, Silvano Garibaldi, Eleonora Bianconi et autres

Long non-coding RNA (lncRNA) may be involved in dysfunction of pulmonary artery endothelial cells (PAEC) and, thus, in pulmonary arterial hypertension (PAH) pathobiology. We screened the RNA expression profile of commercial human PAEC (hPAEC) exposed to increased hydrostatic pressure, and found that …

it, nl (code pays fourni par la source)

5 citations Vascular Pharmacology
2024 conference-abstract OpenAlex

Abstract 4121527: Empagliflozin Improves Mitochondrial Biogenesis in Pulmonary Arterial Hypertension

Keimei Yoshida, Eszter Margit Tóth, Erik Duijvelaar, Beau Neep et autres

Background: Pulmonary arterial hypertension (PAH) is a devastating disease characterized by endothelial proliferation and progressive pulmonary vascular (PV) remodeling. Empagliflozin (EMPA), a sodium glucose cotransporter 2 (SGLT2) inhibitor, benefits heart failure patients, potentially mediated by improved mitochondrial function. This study aimed to …

jp, nl, us (code pays fourni par la source)

0 citations Circulation
Accès ouvert 2024 article OpenAlex

DLC1 promotes mechanotransductive feedback for YAP via RhoGAP-mediated focal adhesion turnover

Aukie Hooglugt, Miesje M. van der Stoel, Apeksha Shapeti, Beau Neep et autres

Angiogenesis is a tightly controlled dynamic process demanding a delicate equilibrium between pro-angiogenic signals and factors that promote vascular stability. The spatiotemporal activation of the transcriptional co-factors YAP (herein referring to YAP1) and TAZ (also known WWTR1), collectively denoted YAP/TAZ, is crucial …

nl, be, de (code pays fourni par la source)

15 citations Journal of Cell Science
2023 conference-abstract OpenAlex

Empagliflozin improves mitochondrial biogenesis and ameliorates vascular remodeling in experimental pulmonary arterial hypertension

Erik Duijvelaar, Keimei Yoshida, Xiao‐Qing Sun, Beau Neep et autres

Introduction: Pulmonary arterial hypertension (PAH) is a devastating disease in which narrowing of the pulmonary vasculature often leads to right ventricular (RV) failure. Empagliflozin (EMPA), a sodium-glucose cotransporter 2 (SGLT2) inhibitor, provides well-established clinical benefits for patients with heart failure. Aims and …

nl, jp (code pays fourni par la source)

1 citation

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