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Profil bibliographique

Tomomi Yamashita

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

6Publications signalées
42Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Stress Responses and CortisolTryptophan and brain disordersNeuroscience and Neuropharmacology ResearchMultiple Sclerosis Research StudiesMitochondrial Function and Pathology

Les publications récentes

Accès ouvert 2025 article OpenAlex

THE EFFICACY OF THE NOVEL 5-HT1A RECEPTOR AGONIST DSP-1181 ON OBSESSIVE-COMPULSIVE DISORDER LIKE BEHAVIOR IN A MOUSE MODEL OF OPTOGENETIC NEURAL MODULATION

Fumiaki Sato, Yusuke Nagano, Daiki Tachibana, Hiroko Ikeda et autres

Abstract Background Obsessive-compulsive disorder (OCD) is a psychiatric disorder characterized by the presence of obsessions and compulsions, leading to significant impairment in social and occupational functioning. The current first-line treatment for OCD involves the use of selective serotonin reuptake inhibitors (SSRIs). However, …

jp (code pays fourni par la source)

0 citations The International Journal of Neuropsychopharmacology
Accès ouvert 2023 article OpenAlex

Metabolic changes in selective brain regions induced by chronic stress in mice and their involvement in emotional disturbance

Kohei Ota, Hirotaka Nagai, Wenran Qiu, Io Horikawa et autres

Chronic stress causes depressed mood and cognitive deficits and predisposes to mental illness. Brain metabolic changes are linked to stress pathology, but their molecular mechanism and functional significance remain unknown. In this study, we exposed C57BL/6N male mice to chronic social defeat …

jp, gb, nz (code pays fourni par la source)

0 citations Proceedings for Annual Meeting of The Japanese Pharmacological Society
Accès ouvert 2022 article OpenAlex

Chronic social stress alters synaptic central metabolism for depression

Hirotaka Nagai, Midori Nagai, Chisato Numa, Tomomi Yamashita et autres

Chronic social stress induces emotional and cognitive disturbances and is a risk for mental illness. Reduced neuronal activity in the medial prefrontal cortex (mPFC) underlies these behavioral abnormalities. However, the subcellular origin and process of this neuronal change remain elusive. Here we …

jp, us, gb (code pays fourni par la source)

0 citations Proceedings for Annual Meeting of The Japanese Pharmacological Society
Accès ouvert 2022 article OpenAlex

Roles of synaptic mitochondrial regulations for stress susceptibility

Chisato Numa, Hirotaka Nagai, Midori Nagai, Tomomi Yamashita et autres

Chronic social stress induces neuronal dysfunctions in the medial prefrontal cortex (mPFC) for emotional and cognitive disturbances. However, the subcellular mechanism remains elusive. Here we examined ultrastructural and multi-omics changes in the mPFC in a mouse model of social defeat stress. Acute …

jp, us, gb (code pays fourni par la source)

0 citations Proceedings for Annual Meeting of The Japanese Pharmacological Society
Accès ouvert 2022 article OpenAlex

Analysis of synaptic structural changes induced by chronic social stress and their molecular mechanisms

Hirotaka Nagai, Midori Nagai, Chisato Numa, Kohei Ota et autres

Excessive or chronic social stress induces emotional and cognitive disturbances and precipitates mental illness. Altered neuronal morphology and functions in the medial prefrontal cortex (mPFC) underlie these behavioral abnormalities. However, its subcellular mechanisms remain elusive. Here we examined ultrastructural and multi-omics changes …

jp, us (code pays fourni par la source)

0 citations Proceedings for Annual Meeting of The Japanese Pharmacological Society
Accès ouvert 2020 article OpenAlex

Benefits of eculizumab in AQP4+ neuromyelitis optica spectrum disorder: Subgroup analyses of the randomized controlled phase 3 PREVENT trial

Jacqueline Palace, Dean M. Wingerchuk, Kazuo Fujihara, Achim Berthele et autres

BACKGROUND: Antibodies to the aquaporin-4 (AQP4) water channel in neuromyelitis optica spectrum disorder (NMOSD) are reported to trigger the complement cascade, which is implicated in astrocyte damage and subsequent neuronal injury. The PREVENT study demonstrated that the terminal complement inhibitor eculizumab reduces …

gb, us, jp, de, es, kr, tr, ru, my, tw (code pays fourni par la source)

42 citations Multiple Sclerosis and Related Disorders

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