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Profil bibliographique

Alina Garbuzov

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

15Publications signalées
879Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Telomeres, Telomerase, and SenescenceSperm and Testicular FunctionTissue Engineering and Regenerative MedicineReproductive Biology and FertilityNeurobiology and Insect Physiology Research

Les publications récentes

Accès ouvert 2024 article OpenAlex

Clonal inactivation of TERT impairs stem cell competition

Kazuteru Hasegawa, Yang Zhao, Alina Garbuzov, M. Ryan Corces et autres

Abstract Telomerase is intimately associated with stem cells and cancer, because it catalytically elongates telomeres—nucleoprotein caps that protect chromosome ends1. Overexpression of telomerase reverse transcriptase (TERT) enhances the proliferation of cells in a telomere-independent manner2–8, but so far, loss-of-function studies have provided …

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18 citations Nature
Accès ouvert 2021 preprint OpenAlex

Clonal inactivation of telomerase promotes accelerated stem cell differentiation

Kazuteru Hasegawa, Yang Zhao, Alina Garbuzov, M. Ryan Corces et autres

Summary Telomerase is intimately associated with stem cells and upregulated in cancer, where it serves essential roles through its catalytic action in elongating telomeres, nucleoprotein caps that protect chromosome ends 1 . Overexpression of the telomerase reverse transcriptase (TERT) enhances cell proliferation …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2018 article OpenAlex

Purification of GFRα1+ and GFRα1– Spermatogonial Stem Cells Reveals a Niche-Dependent Mechanism for Fate Determination

Alina Garbuzov, Matthew F. Pech, Kazuteru Hasegawa, Meena Sukhwani et autres

Undifferentiated spermatogonia comprise a pool of stem cells and progenitor cells that show heterogeneous expression of markers, including the cell surface receptor GFRα1. Technical challenges in isolation of GFRα1+ versus GFRα1- undifferentiated spermatogonia have precluded the comparative molecular characterization of these subpopulations …

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67 citations Stem Cell Reports
2016 article OpenAlex

Mutant nucleophosmin1 (NPM1) Links Protein Translation with Oncogenic Metabolism in Leukemia

Marisa M. Juntilla, Alina Garbuzov, Natalie Ortiz, Julia N. Eberhard et autres

Abstract Nucleophosmin 1 (NPM1) is the most common gene mutated in acute myeloid leukemia (AML). Several putative mechanisms for the role of NPM1 in the generation of AML have been proposed, including acting as a tumor suppressor by promoting the degradation of …

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0 citations Blood
Accès ouvert 2015 preprint OpenAlex

High telomerase is a hallmark of undifferentiated spermatogonia and is required for maintenance of male germline stem cells

Matthew F. Pech, Alina Garbuzov, Kazuteru Hasegawa, Meena Sukhwani et autres

Telomerase inactivation causes loss of the male germline in worms, fish, and mice, indicating a conserved dependence on telomere maintenance in this cell lineage. Here, using telomerase reverse transcriptase (Tert) reporter mice, we found that very high telomerase expression is a hallmark …

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72 citations Genes & Development
2015 conference-abstract OpenAlex

Abstract 980: Encoding immortality: Transcriptional control of telomerase in stem cells in vivo

Matthew F. Pech, Alina Garbuzov, Meena Sukhwani, Bérénice Anath Benayoun et autres

Abstract One of the invariant features of human cancer is unlimited proliferation, a hallmark conferred by telomerase in 90% tumors. Somatic mutations in the telomerase reverse transcriptase (TERT) gene promoter are highly recurrent in human cancers. Telomerase is also critically important in …

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0 citations Cancer Research
Accès ouvert 2015 article OpenAlex

Reversibility of Defective Hematopoiesis Caused by Telomere Shortening in Telomerase Knockout Mice

Aparna Raval, Gregory K. Behbehani, Le Xuan Truong Nguyen, DANIEL W. THOMAS et autres

Telomere shortening is common in bone marrow failure syndromes such as dyskeratosis congenita (DC), aplastic anemia (AA) and myelodysplastic syndromes (MDS). However, improved knowledge of the lineage-specific consequences of telomere erosion and restoration of telomere length in hematopoietic progenitors is required to …

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33 citations PLoS ONE

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