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Profil bibliographique

Melvin M. Evers

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

62Publications signalées
2256Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Genetic Neurodegenerative DiseasesMuscle Physiology and DisordersRNA Research and SplicingCRISPR and Genetic EngineeringMitochondrial Function and Pathology

Les publications récentes

Accès ouvert 2024 article OpenAlex

Preclinical efficacy and safety of adeno-associated virus 5 alpha-galactosidase: A gene therapy for Fabry disease

Jolanda M.P. Liefhebber, Giso Brasser, Elisabeth A. Spronck, Roelof Ottenhoff et autres

We developed a novel adeno-associated virus 5 gene therapy (AAV5-GLA) expressing human alpha-galactosidase A (GLA) under the control of a novel, small and strong, liver-restricted promoter. We assessed the preclinical potential of AAV5-GLA for treating Fabry disease, an X-linked hereditary metabolic disorder …

nl (code pays fourni par la source)

9 citations Molecular Therapy — Methods & Clinical Development
Accès ouvert 2024 conference-abstract OpenAlex

A006 Splicing dysregulation is a common theme across Huntington’s disease mouse models and can be rescued by huntingtin lowering

Leontien van der Bent, Melvin M. Evers, Astrid Vallès

Huntington’s disease (HD) exhibits a complex molecular phenotype, including significant transcriptional and splicing dysregulation. While splicing dysregulation is known to play a critical role in other repeat expansion disorders such as myotonic dystrophy type 1, its conservation across HD models remains to …

nl (code pays fourni par la source)

0 citations
Accès ouvert 2024 article OpenAlex

Exon 1-targeting miRNA reduces the pathogenic exon 1 HTT protein in Huntington's disease models

Marina Sogorb-González, Christian Landles, Nicholas S. Caron, Anouk Stam et autres

Huntington's disease (HD) is a fatal neurodegenerative disease caused by a trinucleotide repeat expansion in exon 1 of the huntingtin gene (HTT) that results in toxic gain of function and cell death. Despite its monogenic cause, the pathogenesis of HD is highly …

nl, gb, ca, us (code pays fourni par la source)

27 citations Brain
Accès ouvert 2024 article OpenAlex

LinQURE: A novel AAV gene silencing platform that supports multi-transcript targeting for complex disorders

Irena Bočkaj, Anna Moreno Garcia, Pablo de Miguel Herraiz, Sonay Keskin et autres

, allowing more efficient downregulation of their disease-causing mRNA targets. This approach supports the development of multi-targeting therapeutic strategies, enabling gene therapy products to adapt to more complex multigenic indications, thus expanding the toolbox of readily available gene therapies.

nl (code pays fourni par la source)

0 citations Molecular Therapy — Nucleic Acids
Accès ouvert 2022 article OpenAlex

AAV5-miHTT-mediated huntingtin lowering improves brain health in a Huntington’s disease mouse model

Sarah B. Thomson, Anouk Stam, Cynthia C. Brouwers, Valentina Fodale et autres

Huntingtin (HTT)-lowering therapies show great promise in treating Huntington's disease. We have developed a microRNA targeting human HTT that is delivered in an adeno-associated serotype 5 viral vector (AAV5-miHTT), and here use animal behaviour, MRI, non-invasive proton magnetic resonance spectroscopy and striatal …

ca, nl, it, us (code pays fourni par la source)

26 citations Brain
Accès ouvert 2022 article OpenAlex

Enterovirus D68 Infection in Human Primary Airway and Brain Organoids: No Additional Role for Heparan Sulfate Binding for Neurotropism

Adithya Sridhar, Josse A. Depla, Lance Mulder, Eveliina Karelehto et autres

Recent outbreaks of enterovirus D68, a nonpolio enterovirus, is associated with a serious neurological condition in young children, acute flaccid myelitis (AFM). As there is no antiviral treatment or vaccine available for EV-D68 it is important to better understand how EV-D68 causes …

nl (code pays fourni par la source)

32 citations Microbiology Spectrum
2022 conference-abstract OpenAlex

I04 Aav5-mihtt gene therapy mediates sustained mutant huntingtin lowering in brain and cerebrospinal fluid of Huntington disease minipigs up to 4 years

Leontien van der Bent, Cynthia C. Brouwers, Robin Pocornie, Anouk Stam et autres

Background Huntingtin (HTT)-lowering gene therapies are being evaluated in clinical trials. We have previously shown that one-time intrastriatal administration of an adeno-associated viral vector serotype 5 encoding an engineered miRNA (AAV5-miHTT) targeting human mutant huntingtin (mHTT) protein leads to sustained CSF mHTT …

nl, cz (code pays fourni par la source)

0 citations
Accès ouvert 2022 article OpenAlex

Emerging Therapies for Huntington’s Disease – Focus on N-Terminal Huntingtin and Huntingtin Exon 1

M. Leontien van der Bent, Melvin M. Evers, Astrid Vallès

Abstract: Huntington’s disease is a devastating heritable neurodegenerative disorder that is caused by the presence of a trinucleotide CAG repeat expansion in the Huntingtin gene, leading to a polyglutamine tract in the protein. Various mechanisms lead to the production of N-terminal Huntingtin …

nl (code pays fourni par la source)

16 citations Biologics
Accès ouvert 2022 article OpenAlex

Human Brain Organoids as Models for Central Nervous System Viral Infection

Josse A. Depla, Lance Mulder, Renata Vieira de Sá, Morgane Wartel et autres

Pathogenesis of viral infections of the central nervous system (CNS) is poorly understood, and this is partly due to the limitations of currently used preclinical models. Brain organoid models can overcome some of these limitations, as they are generated from human derived …

nl (code pays fourni par la source)

60 citations Viruses

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