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Profil bibliographique

Paul R. Mark

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

48Publications signalées
2081Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Genomics and Rare DiseasesGenomic variations and chromosomal abnormalitiesGenetics and Neurodevelopmental DisordersEpigenetics and DNA MethylationConnective tissue disorders research

Les publications récentes

Accès ouvert 2026 article OpenAlex

A three generation family with VACTERL association is found to have a rare form of diamond-blackfan anaemia

Iryna Leshchyns’ka, Debjani Das, Victoria C. O׳Reilly, Alena Sipka et autres

The spectrum of congenital malformations in VACTERL association varies among patients and can be differentially diagnosed with CHARGE syndrome, Fanconi anaemia, and others (reviewed in Solomon 2011). Despite overlapping clinical findings, the genetic causes of these diseases are distinct. In this context, …

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0 citations European Journal of Human Genetics
Accès ouvert 2025 article OpenAlex

A zebrafish model of nicotinamide adenine dinucleotide (NAD+) deficiency-derived congenital disorders

Visakuo Tsurho, Carla Gilliland, Jessica Ensing, Elizabeth VanSickle et autres

Developmental NAD + deficiency is associated with diverse congenital malformations. Congenital NAD deficiency disorder (CNDD) is a multisystem developmental condition characterized by cardiac, renal, vertebral, and limb anomalies, among others. It is caused by biallelic pathogenic variants in genes involved in the …

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0 citations Developmental Biology
Accès ouvert 2025 preprint OpenAlex

A zebrafish model of nicotinamide adenine dinucleotide (NAD + ) deficiency-derived congenital disorders

Visakuo Tsurho, Carla Gilliland, Jessica Ensing, Elizabeth VanSickle et autres

Abstract Congenital NAD deficiency disorder (CNDD) is a multisystem condition in which cardiac, renal, vertebral, and limb anomalies are most common, but anomalies in all organ systems have been identified. Patients with this condition have biallelic pathogenic variants involving genes in the …

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0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2024 article OpenAlex

Heterozygous loss-of-function variants in DOCK4 cause neurodevelopmental delay and microcephaly

Charlotte Herbst, Viktoria Bothe, Meret Wegler, Susanne Axer-Schaefer et autres

Neurons form the basic anatomical and functional structure of the nervous system, and defects in neuronal differentiation or formation of neurites are associated with various psychiatric and neurodevelopmental disorders. Dynamic changes in the cytoskeleton are essential for this process, which is, inter …

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6 citations Human Genetics
Accès ouvert 2024 article OpenAlex

P330: A novel WARS2 variant impacting dimerization in trans with a recurrent pathogenic variant in a child with features of NEMMLAS

Christine Bergeon Burns, Elizabeth VanSickle, Paul R. Mark, Jeremy W. Prokop

Biallelic pathogenic variants in mitochondrial tryptophanyl tRNA synthetase 2 (WARS2; HGNC:12730) cause neurodevelopmental disorder, mitochondrial, with abnormal movements and lactic acidosis, with or without seizures (NEMMLAS; OMIM 617710). First reported in 2017, 31 patients with WARS2-related disorder have been described. Features include …

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0 citations Genetics in Medicine Open
Accès ouvert 2023 preprint OpenAlex

Heterozygous loss-of function variants in DOCK4 cause neurodevelopmental delay and microcephaly

Charlotte Herbst, Viktoria Bothe, Meret Wegler, Susanne Axer-Schaefer et autres

Abstract Neurons form the basic anatomical and functional structure of the nervous system, and defects in neuronal differentiation or formation of neurites are associated with various psychiatric and neurodevelopmental disorders. Dynamic changes in the cytoskeleton are essential for this process, which is, …

us, de, fr, au, il, kr (code pays fourni par la source)

0 citations Research Square
Accès ouvert 2023 article OpenAlex

Gain and loss of function variants in EZH1 disrupt neurogenesis and cause dominant and recessive neurodevelopmental disorders

Carolina Gracia-Diaz, Yijing Zhou, Qian Yang, Reza Maroofian et autres

Genetic variants in chromatin regulators are frequently found in neurodevelopmental disorders, but their effect in disease etiology is rarely determined. Here, we uncover and functionally define pathogenic variants in the chromatin modifier EZH1 as the cause of dominant and recessive neurodevelopmental disorders …

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27 citations Nature Communications
Accès ouvert 2022 article OpenAlex

Gain-of-function mutations in KCNK3 cause a developmental disorder with sleep apnea

Janina Sörmann, Marcus Schewe, Peter Proks, Thibault R. H. Jouen-Tachoire et autres

Abstract Sleep apnea is a common disorder that represents a global public health burden. KCNK3 encodes TASK-1, a K + channel implicated in the control of breathing, but its link with sleep apnea remains poorly understood. Here we describe a new developmental …

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34 citations Nature Genetics
Accès ouvert 2022 preprint OpenAlex

Autosomal recessive LRP1-related syndrome featuring cardiopulmonary dysfunction, bone dysmorphology, and corneal clouding.

Paul R. Mark, Stephen A. Murray, Tao Yang, Alexandra Eby et autres

analysis using multiple human/mouse data sets reveals a correlation to patient phenotypes of Peters plus syndrome with additional severe cardiomyopathy and blood vessel development complications linked to neural crest cells.

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7 citations PubMed
2022 article OpenAlex

Viewing teratogens through the lens of nicotinamide adenine dinucleotide ( NAD +)

Paul R. Mark, Sally L. Dunwoodie

BACKGROUND: Nicotinamide adenine dinucleotide (NAD+) depletion is associated with numerous diseases in humans. Recently it was revealed that genetic blockage of the NAD+ synthesis pathway in humans causes birth defects in multiple organ systems and miscarriage. Additionally, mice with NAD+ deficiency created …

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6 citations Birth Defects Research
Accès ouvert 2022 article OpenAlex

Genomic and phenotypic characterization of 404 individuals with neurodevelopmental disorders caused by CTNNB1 variants

Sayaka Kayumi, Luis Alberto Pérez-Jurado, María Palomares‐Bralo, Sneha A. Rangu et autres

PURPOSE: Germline loss-of-function variants in CTNNB1 cause neurodevelopmental disorder with spastic diplegia and visual defects (NEDSDV; OMIM 615075) and are the most frequent, recurrent monogenic cause of cerebral palsy (CP). We investigated the range of clinical phenotypes owing to disruptions of CTNNB1 …

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50 citations Genetics in Medicine
Accès ouvert 2022 preprint OpenAlex

Gain and loss of function variants in EZH1 disrupt neurogenesis timing and cause overlapping neurodevelopmental disorders

Carolina Gracia-Diaz, Yijing Zhou, Qian Yang, Chul‐Hwan Lee et autres

Abstract Genetic disruption of chromatin regulators is frequently found in neurodevelopmental disorders (NDDs). While chromatin regulators are attractive therapeutic targets, studies to determine their implication in the etiology of NDDs are limited, preventing advances in diagnosis and treatment strategies. Here, we uncover …

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1 citation medRxiv

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