Yoshihisa Kobayashi

Mutation of CMTR2 in Lung Adenocarcinoma Alters RNA Alternative Splicing and Reveals Therapeutic Vulnerabilities

Shigenari Nukaga, Kouya Shiraishi, Kenta Hamabe, Akifumi Mochizuki et autres

jp, gb (code pays fourni par la source)

Efficacy of Conventional and Novel Tyrosine Kinase Inhibitors for Uncommon EGFR Mutations—An In Vitro Study

Hana Oiki, Kenichi Suda, Akira Hamada, Toshio Fujino et autres

jp, us (code pays fourni par la source)

Efficacy of Novel Third-Generation Tyrosine Kinase Inhibitors for Uncommon EGFR Mutations – an In Vitro Study

Hana Oiki, Kenichi Suda, Akira Hamada, Toshio Fujino et autres

jp, gb (code pays fourni par la source)

Systematically developing a registry of splice-site creating variants utilizing massive publicly available transcriptome sequence data

Naoko Iida, Ai Okada, Yoshihisa Kobayashi, Kenichi Chiba et autres

us (code pays fourni par la source)

RNA sensing induced by chromosome missegregation augments anti-tumor immunity

Nobunari Sasaki, Mizuki Homme, Takahiko Murayama, Tatsuya Osaki et autres

jp, us, au, gb (code pays fourni par la source)

Systematically developing a registry of splice-site creating variants utilizing massive publicly available transcriptome sequence data

Naoko Iida, Ai Okada, Yoshihisa Kobayashi, Kenichi Chiba et autres

Supplementary Table 1 from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres

Supplementary Figure 1A from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres

Supplementary Figure 1B from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres

Supplementary Figure 2 from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres

Supplementary Figure Legend from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres

Supplementary Figure 1A from Characterization of EGFR T790M, L792F, and C797S Mutations as Mechanisms of Acquired Resistance to Afatinib in Lung Cancer

Yoshihisa Kobayashi, Koichi Azuma, Hiroki Nagai, Young Hak Kim et autres