Profil bibliographique

Patrick Paul Torreiter

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

3Publications signalées

4Citations signalées

1Affiliations récentes

Les institutions déclarées

Les domaines associés

Vector-borne infectious diseasesIron Metabolism and DisordersHemoglobinopathies and Related DisordersErythropoietin and Anemia TreatmentBlood groups and transfusion

Les publications récentes

Accès ouvert 2026 article OpenAlex

Regional and demographic patterns of seroprevalence of Borrelia burgdorferi IgG and IgM antibodies in blood donors in Austria

Mateusz Markowicz, Andreas Wolfsbauer, Christof Jungbauer, Patrick Paul Torreiter et autres

Knowledge about the epidemiology of Lyme borreliosis in Austria remains limited. We conducted a nationwide cross-sectional study among blood donors from all Austrian provinces to assess the exposure to Borrelia burgorferi sensu lato. A total of 1841 serum samples from blood donors …

at (code pays fourni par la source)

0 citations Ticks and Tick-borne Diseases

Accès ouvert 2022 article OpenAlex

Pilot Study to Gain First Indications for the Impact of a 3-Month’s Oral Intake of a Sucrosomial Iron Supplement on Hemoglobin in Iron-Deficient Blood Donors

Patrick Paul Torreiter, Camilla Drexler, Wolfgang Schimetta, Petra Krakowitzky et autres

Introduction: Regular whole blood donors often suffer from iron deficiency (ID) or iron deficiency anemia due to the loss of 200–300 mg of iron with each donation. Hemoglobin (Hb) as donor eligibility criterion reflects iron stores only poorly. ID in blood donors …

at (code pays fourni par la source)

1 citation Transfusion Medicine and Hemotherapy

Accès ouvert 2021 article OpenAlex

Ethical Issues and Management of Fetal Hemolytic Anemia Caused by Anti-Rh17 in a Multipara with Rare –D– Phenotype

Patrick Paul Torreiter, Susanne Macher, Eva-Maria Matzhold, Bernhard Resch et autres

BACKGROUND: The development of allo-anti-Rh17 (anti-Hr0) in a -D- phenotype whose red blood cells (RBCs) lack CcEe antigens is most likely triggered by transfusion, transplantation, or pregnancy. Gene conversion is the predominating factor in generating RHD-CE-D and RHCE-D-CE hybrids like -D-. METHODS: …

at (code pays fourni par la source)

3 citations Transfusion Medicine and Hemotherapy

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