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Profil bibliographique

Nicole van Vliet

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

37Publications signalées
1123Citations signalées
1Affiliations récentes

Les institutions déclarées

Les domaines associés

Connective tissue disorders researchAortic aneurysm repair treatmentsDNA Repair MechanismsAortic Disease and Treatment ApproachesGenetics, Aging, and Longevity in Model Organisms

Les publications récentes

Accès ouvert 2026 preprint OpenAlex

Septins promote breast cancer cell invasion in 3D collagen gels by influencing actin-based protrusion formation

Anouk van der Net, Klara Beslmüller, Nicole van Vliet, Margherita Tavasso et autres

Summary Septins are cytoskeletal proteins that contribute to essential cellular processes such as cell migration and cell division through interactions with the cell membrane and the cytoskeleton. High expression of septins is correlated with breast cancer malignancy and promotes cell invasion, but …

nl (code pays fourni par la source)

0 citations bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 article OpenAlex

A Del(5 Ncf1 - Fkbp6 ) mouse model of Williams syndrome shows coronary, aortic, and cerebral vascular abnormalities with behavioral deficits

Laurens W. J. Bosman, Hamid el Azzouzi, Lieke Kros, Yanto Ridwan et autres

Abstract Williams syndrome is a developmental disorder caused by a microdeletion entailing the loss of a single copy of 25–27 genes on chromosome 7q11.23. Patients suffer from cardiovascular and neuropsychological symptoms. Structural abnormalities of the cardiovascular system in Williams syndrome have been …

nl, cn (code pays fourni par la source)

1 citation PNAS Nexus
Accès ouvert 2025 article OpenAlex

Plectin affects cell viscoelasticity at small and large deformations

James P. Conboy, Mathilde G Lettinga, Nicole van Vliet, Lilli Winter et autres

cells, suggesting a more sparse cytoskeletal network. Confocal imaging indicated that this was due to a marked change in the architecture of the vimentin network, from a fine meshwork in wild-type cells to a bundled network in the plectin knockout cells. Our …

nl, at, us (code pays fourni par la source)

1 citation Biophysical Journal
Accès ouvert 2025 preprint OpenAlex

Plectin affects cell viscoelasticity at small and large deformations

James P. Conboy, Mathilde G Lettinga, Nicole van Vliet, Lilli Winter et autres

ABSTRACT Plectin is a giant protein of the plakin family that crosslinks the cytoskeleton of mammalian cells. It is expressed in virtually all tissues and its dysfunction is associated with various diseases such as skin blistering. There is evidence that plectin regulates …

nl, at, us (code pays fourni par la source)

1 citation bioRxiv (Cold Spring Harbor Laboratory)
Accès ouvert 2025 article OpenAlex

Dietary Restriction Mitigates Vascular Aging, Modulates the cGAS ‐ STING Pathway and Reverses Macrophage‐Like VSMC Phenotypes in Progeroid DNA ‐Repair‐Deficient Ercc1 Δ /− Mice

Sanne J. M. Stefens, Janette van der Linden, José María Heredia‐Genestar, Renata M. C. Brandt et autres

ABSTRACT Aging is a major risk factor for cardiovascular diseases, and the accumulation of DNA damage significantly contributes to the aging process. This study aimed to identify the underlying molecular mechanisms of vascular aging in DNA‐repair‐deficient progeroid Ercc1 Δ/− mice and to …

nl, de (code pays fourni par la source)

4 citations Aging Cell
Accès ouvert 2025 article OpenAlex

Induction of cardiac fibulin-4 protects against pressure overload-induced cardiac hypertrophy and heart failure

Elza D. van Deel, Matthijs Snelders, Nicole van Vliet, Luuk te Riet et autres

Abstract The prevailing view of fibulin-4 deficient mice is that the cardiac phenotype is the result of aortic and/or valvular disease. In the present study, we have tested whether the cardiac phenotype is, at least in part, the consequence of primary cardiac …

nl, gb, us, jp, ca (code pays fourni par la source)

1 citation Communications Biology
Accès ouvert 2024 article OpenAlex

Increased vascular smooth muscle cell senescence in aneurysmal Fibulin-4 mutant mice

Sanne J. M. Stefens, Nicole van Vliet, Arne IJpma, Joyce Burger et autres

Abstract Aortic aneurysms are dilatations of the aorta that can rupture when left untreated. We used the aneurysmal Fibulin-4R/R mouse model to further unravel the underlying mechanisms of aneurysm formation. RNA sequencing of 3-month-old Fibulin-4R/R aortas revealed significant upregulation of senescence-associated secretory …

nl (code pays fourni par la source)

10 citations npj Aging
Accès ouvert 2024 article OpenAlex

Hybrid Molecular and Functional Micro-CT Imaging Reveals Increased Myocardial Apoptosis Preceding Cardiac Failure in Progeroid Ercc1 Mice

Bibi S. van Thiel, Martine de Boer, Yanto Ridwan, Marion G. J. de Kleijnen et autres

PURPOSE: In this study, we explored the role of apoptosis as a potential biomarker for cardiac failure using functional micro-CT and fluorescence molecular tomography (FMT) imaging techniques in Ercc1 mutant mice. Ercc1 is involved in multiple DNA repair pathways, and its mutations …

nl, de (code pays fourni par la source)

2 citations Molecular Imaging and Biology
Accès ouvert 2024 article OpenAlex

Ercc1 DNA repair deficiency results in vascular aging characterized by VSMC phenotype switching, ECM remodeling, and an increased stress response

Janette van der Linden, Sanne J. M. Stefens, José María Heredia‐Genestar, Yanto Ridwan et autres

Abstract Cardiovascular diseases are the number one cause of death globally. The most important determinant of cardiovascular health is a person's age. Aging results in structural changes and functional decline of the cardiovascular system. DNA damage is an important contributor to the …

nl, us (code pays fourni par la source)

25 citations Aging Cell
Accès ouvert 2023 preprint OpenAlex

A long-deletion mouse model of Williams syndrome reveals Ncf1 -dependent modulation of vascular and neural phenotypes

Laurens W. J. Bosman, Lieke Kros, Nicole van Vliet, Yanto Ridwan et autres

Abstract Williams syndrome is a developmental disorder caused by a microdeletion entailing the loss of a single copy of 25-27 genes on chromosome 7q11.23. Patients suffer from cardiovascular and neuropsychological symptoms. Structural abnormalities of the cardiovascular system in Williams syndrome have been …

nl, cn, gb (code pays fourni par la source)

2 citations bioRxiv (Cold Spring Harbor Laboratory)
2022 conference-abstract OpenAlex

Abstract 11841: Activation of Cardiac Fibulin-4 Protects Against Pressure Overload-Induced Cardiac Hypertrophy and Heart Failure

Elza van Deel, Nicole van Vliet, Thierry van den Bosch, Ariane van Spreeuwel et autres

Cutis laxa is a rare disorder caused by mutations in the extracellular matrix (ECM) protein Fibulin-4 and patients are susceptible to stress-induced cardiac dysfunction. Mice with a 75% reduction in Fibulin-4 expression (Fibulin-4 R/R ) developed cardiac hypertrophy, dilation and dysfunction as …

nl, jp (code pays fourni par la source)

0 citations Circulation

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