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Profil bibliographique

Zhefang Wang

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

35Publications signalées
1396Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Pancreatic and Hepatic Oncology ResearchEpigenetics and DNA MethylationCancer Cells and MetastasisRNA modifications and cancerCancer, Hypoxia, and Metabolism

Les publications récentes

Accès ouvert 2026 article OpenAlex

LSD1-GLS2 axis drives subtype-specific chemoresistance in pancreatic cancer through glutaminolysis reprogramming

Zhefang Wang, Qiu Huang, Jiangang Zhao, Zicheng Lyu et autres

Pancreatic ductal adenocarcinoma (PDAC) remains a highly lethal malignancy due to its aggressive biology and therapeutic resistance. Lysine-specific demethylase 1 (LSD1), an epigenetic regulator, is overexpressed in PDAC and linked to poor prognosis, yet its context-dependent roles in metabolic subtypes and chemoresistance …

de, cn (code pays fourni par la source)

0 citations Cell Death and Disease
Accès ouvert 2026 article OpenAlex

Targeting the CAF-GDF15 axis attenuates AKT-mediated mitochondrial rewiring and chemoradiation resistance in esophageal adenocarcinoma

Ningbo Fan, Pinwei Deng, Zhefang Wang, Feng Ju et autres

BACKGROUND: Therapeutic resistance in esophageal adenocarcinoma (EAC) remains poorly understood at the level of tumor-stroma interactions. We investigated whether cancer-associated fibroblast (CAF)-derived signaling contributes to chemoradiation resistance and adverse clinical outcomes in EAC, with a focus on the functional role of GDF15 …

cn, de, nl (code pays fourni par la source)

0 citations Journal of Experimental & Clinical Cancer Research
Accès ouvert 2026 other OpenAlex

Targeting the CAF-GDF15 axis attenuates AKT-mediated mitochondrial rewiring and chemoradiation resistance in esophageal adenocarcinoma

Ningbo Fan, Pinwei Deng, Zhefang Wang, J. Feng et autres

Abstract Background Therapeutic resistance in esophageal adenocarcinoma (EAC) remains poorly understood at the level of tumor-stroma interactions. We investigated whether cancer-associated fibroblast (CAF)-derived signaling contributes to chemoradiation resistance and adverse clinical outcomes in EAC, with a focus on the functional role of …

cn, de, nl (code pays fourni par la source)

0 citations Figshare
Accès ouvert 2026 other OpenAlex

Targeting the CAF-GDF15 axis attenuates AKT-mediated mitochondrial rewiring and chemoradiation resistance in esophageal adenocarcinoma

Ningbo Fan, Pinwei Deng, Zhefang Wang, J. Feng et autres

Abstract Background Therapeutic resistance in esophageal adenocarcinoma (EAC) remains poorly understood at the level of tumor-stroma interactions. We investigated whether cancer-associated fibroblast (CAF)-derived signaling contributes to chemoradiation resistance and adverse clinical outcomes in EAC, with a focus on the functional role of …

cn, de, nl (code pays fourni par la source)

0 citations Figshare
Accès ouvert 2026 article OpenAlex

Single-cell profiling of PBMCS reveals an immune signature of irAEs in anti-PD-1-treated acral melanoma patients

Qi Bao, Jie Pan, Zhefang Wang

Introduction: Immune checkpoint inhibitors (ICIs) targeting PD-1 have revolutionized melanoma treatment, yet their clinical efficacy is frequently limited by immune-related adverse events (irAEs). The underlying mechanisms of irAEs remain poorly defined, particularly in the acral melanoma subtype. Methods: To identify peripheral immune …

cn (code pays fourni par la source)

0 citations Frontiers in Immunology
Accès ouvert 2026 article OpenAlex

Mitophagy in pancreatic cancer: mechanistic insights and implications for novel therapeutic strategies

Zhefang Wang, Zicheng Lyu, Raphael Palmen, Qi Bao et autres

Pancreatic ductal adenocarcinoma (PDAC) presents significant treatment challenges, primarily due to its propensity for developing resistance to therapeutic interventions. While the underlying mechanisms remain elusive, they are closely associated with mitochondrial adaptation in response to treatment. Mitophagy, a selective subtype of autophagy …

de, cn (code pays fourni par la source)

1 citation Cell Death Discovery
Accès ouvert 2025 article OpenAlex

AKR1C3 enhances radioresistance in esophageal adenocarcinoma via inhibiting ferroptosis through suppressing TRIM21-mediated ubiquitination of HSPA5

Feng Ju, Jialei Weng, Ningbo Fan, Zhefang Wang et autres

Esophageal adenocarcinoma (EAC) is the predominant subtype of esophageal cancer (EC) in high-income countries, and radioresistance is one of the key factors for the poor prognosis. In this study, we successfully established a radioresistant EAC in vitro model. Aldo-keto reductase 1C3 (AKR1C3) …

de, cn (code pays fourni par la source)

14 citations Cell Death and Disease
Accès ouvert 2025 article OpenAlex

DDX5 super-enhancer promotes vasculogenic mimicry formation and metastasis in nasopharyngeal carcinoma by enhancing ADAM10 transcription

Tian Xia, Haimeng Yin, Qingwen Zhu, Kaiwen Zhang et autres

Anti-angiogenic therapies (AATs) exhibit limited efficacy, as most patients with cancer inevitably develop resistance to them. In this study, data generated using a nasopharyngeal carcinoma orthotopic mouse model, combined with clinical data, reveal compensatory vasculogenic mimicry (VM) formation during AAT treatment and …

cn (code pays fourni par la source)

3 citations Cell Reports Medicine
Accès ouvert 2025 article OpenAlex

IGF2BP1-HAX-1 positive feedback loop-mediated HAX-1 overexpression blocks autophagic flux and promotes chemoresistance in nasopharyngeal carcinoma

Siyu Zhang, Miao Gu, Haimeng Yin, Si Pan et autres

Autophagy is associated with chemoresistance, which is the leading cause of failure in chemotherapeutic treatments. Among the various aspects of autophagy, autophagic flux serves as a critical indicator for evaluating the dynamic processes involved.We report herein that the multifunctional protein HAX-1 promotes …

cn, de (code pays fourni par la source)

9 citations Cellular and Molecular Life Sciences
Accès ouvert 2023 article OpenAlex

Oncogenic role and target properties of the lysine-specific demethylase KDM1A in chronic lymphocytic leukemia

Qu Jiang, Johanna Stachelscheid, Johannes Bloehdorn, Alicja Pacholewska et autres

In chronic lymphocytic leukemia (CLL), epigenetic alterations are considered to centrally shape the transcriptional signatures that drive disease evolution and underlie its biological and clinical subsets. Characterizations of epigenetic regulators, particularly histone-modifying enzymes, are very rudimentary in CLL. In efforts to establish …

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11 citations Blood

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