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Profil bibliographique

R. Zeiser

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

3Publications signalées
15Citations signalées
2Affiliations récentes

Les institutions déclarées

Les domaines associés

Ferroptosis and cancer prognosisImmunotherapy and Immune ResponsesRNA Interference and Gene DeliveryAntimicrobial Peptides and ActivitiesEndoplasmic Reticulum Stress and Disease

Les publications récentes

Accès ouvert 2026 article OpenAlex

Activated ATF6α is a hepatic tumour driver restricting immunosurveillance

Xin Li, Cynthia Lebeaupin, Aikaterini Kadianaki, Clementine Druelle-Cedano et autres

Abstract Hepatocellular carcinoma (HCC) is the fastest growing cause of cancer-related mortality and there are limited therapies 1 . Although endoplasmic reticulum (ER) stress and the unfolded protein response (UPR) are implicated in HCC, the involvement of the UPR transducer ATF6α remains …

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8 citations Nature
Accès ouvert 2026 article OpenAlex

Vertical RAS pathway inhibition in pancreatic cancer drives therapeutically exploitable mitochondrial alterations

Philipp Hafner, S. Keller, Xun Chen, Asma Alrawashdeh et autres

Oncogenic KRAS mutations drive metabolic reprogramming in pancreatic ductal adenocarcinoma (PDAC). Src-homology 2 domain-containing phosphatase 2 (SHP2) is essential for full KRAS activity, and promising dual SHP2/mitogen-activated protein kinase (MAPK) inhibition is currently being tested in clinical trials. Exploitable metabolic adaptations may …

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7 citations Signal Transduction and Targeted Therapy
Accès ouvert 2022 article OpenAlex

S230: HUMAN ΒETA-DEFENSIN 2 TREATMENT MODULATES THE INTESTINAL MICROBIOME AND THE ALLOGENEIC T CELL RESPONSE TO LIMIT ACUTE GRAFT-VERSUS-HOST DISEASE

T. Rückert, G. Andrieux, M. Boerries, N. M. Woessner et autres

Background: Allogeneic hematopoietic cell transplantation (allo-HCT) is a curative therapy option for patients with hematological malignancies. Acute graft-versus-host disease (GVHD) is primarily driven by allogeneic donor T cells and remains a serious cause of morbidity and mortality post allo-HCT. In particular, acute …

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0 citations HemaSphere

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