Aller au contenu principal
2017 article

Niche partitioning and differential transmission of infectious agents

0Citations signalées, ce qui n’est pas une note de qualité
0Institutions déclarées
0Pays d’affiliation déclarés

Le résumé fourni par la source

The transmission potential of an infection, usually measured via the basic reproduction ratio R 0 , is determined by three main factors: (i) duration of latent and infective periods (ii) probability of interaction (relevant to the transmission pathway) between an infective and a susceptible and (iii) probability of transmission when such interaction occurs. While (i) and (ii) can be determined experimentally and through behavioural observations, (iii) is difficult to establish by direct means and is typically estimated via mathematical modelling and observable data, e.g. infection prevalence. A common assumption in such procedures is that the transmissibility of infection is constant across physiologically similar hosts and minor variations of the infective agent. This assumption is not always true. For example, the results in [1] revealed that despite physiological similarities between hosts the transmissibility of the Bartonella bacterium differed markedly. In this study we investigate how different variants of the same infectious agent may have differential transmissibility, and therefore variable transmission potential, due to niche partitioning within the host. In the field of medical infectious disease research there are numerous of examples of pathogens that display tissue tropism, analogous to the ecological concept of niche partitioning. These include HIV, a viral agent that is transmitted horizontally (either sexually or through blood) and Trypanosoma cruzi , the vector-borne agent for Chagas disease which may be transmitted horizontally (eg. through blood transfusions) or congenitally, with the vertically transmitted pathogen persisting after child birth. Here we consider infectious diseases of animals for which there is some evidence of tissue tropism, and devise a preliminary mathematical model to investigate the downstream effects of niche partitioning on pathogen transmission.

Ce résumé expose les affirmations des auteurs. BNTIC ne l’interprète pas comme une validation indépendante des résultats.

Le contrôle bibliographique ouvert

Aucun DOI disponible pour le contrôle Crossref.

Les sujets associés

Mathematical and Theoretical Epidemiology and Ecology ModelsEvolution and Genetic DynamicsViral Infections and Vectors

BNTIC News n’est pas le producteur de ces données. Les publications sont interrogées à la demande dans Crossref, OpenAIRE, DOAJ, Europe PMC, HAL, DataCite, AfricArXiv, ROR et la Banque mondiale, sans clé d’accès. OpenAlex reste optionnel. Aucun service payant n’est nécessaire et aucune donnée externe n’est enregistrée en base. Consulter les sources et leurs limites.