Profil bibliographique

Atsuo Kuramasu

Informations fournies par OpenAlex. Research Africa ne déduit ni nationalité, ni poste, ni coordonnées personnelles.

106Publications signalées
2147Citations signalées
1Affiliations récentes

Les institutions déclarées

SHOWA Medical University

Les domaines associés

Mast cells and histamineCancer Immunotherapy and BiomarkersReceptor Mechanisms and SignalingImmune Cell Function and InteractionCAR-T cell therapy research

Les publications récentes

2026 conference-abstract OpenAlex

Longitudinal analysis of PD-1 receptor occupancy in spleen and tumor-infiltrating lymphocytes in a murine model.

Toshiaki Tsurui, Masahiro Hosonuma, Eiji Funayama, Midori Shida et autres

e14580 Background: Anti-PD-1 antibodies restore T-cell-mediated anti-tumor immune responses by blocking the interaction between PD-1 and programmed cell death ligand 1 (PD-L1). Following anti-PD-1 administration, PD-1 receptor occupancy (RO) on circulating T cells has been shown to vary widely in a dose-independent …

jp, us (code pays fourni par la source)

0 citations Journal of Clinical Oncology
Accès ouvert 2026 article OpenAlex

Cetirizine suppresses cancer-associated fibroblast-mediated fibrotic remodeling and epithelial–mesenchymal transition via histamine H1 receptor inverse agonism

Aya Sasaki, Masahiro Hosonuma, Yuki Maruyama, Eiji Funayama et autres

BACKGROUND: Triple-negative breast cancer (TNBC) is an aggressive subtype characterized by high metastatic potential and limited therapeutic options. Cancer-associated fibroblasts (CAFs) promote epithelial–mesenchymal transition (EMT) and metastatic progression through extracellular matrix (ECM) remodeling and TGF-β signaling; however, the role of histamine H1 …

jp (code pays fourni par la source)

0 citations Breast Cancer Research
Accès ouvert 2026 other OpenAlex

Cetirizine suppresses cancer-associated fibroblast-mediated fibrotic remodeling and epithelial–mesenchymal transition via histamine H1 receptor inverse agonism

Aya Sasaki, Masahiro Hosonuma, Yuki Maruyama, Eiji Funayama et autres

Abstract Background Triple-negative breast cancer (TNBC) is an aggressive subtype characterized by high metastatic potential and limited therapeutic options. Cancer-associated fibroblasts (CAFs) promote epithelial–mesenchymal transition (EMT) and metastatic progression through extracellular matrix (ECM) remodeling and TGF-β signaling; however, the role of histamine …

jp (code pays fourni par la source)

0 citations Figshare
Accès ouvert 2026 other OpenAlex

Cetirizine suppresses cancer-associated fibroblast-mediated fibrotic remodeling and epithelial–mesenchymal transition via histamine H1 receptor inverse agonism

Aya Sasaki, Masahiro Hosonuma, Yuki Maruyama, Eiji Funayama et autres

Abstract Background Triple-negative breast cancer (TNBC) is an aggressive subtype characterized by high metastatic potential and limited therapeutic options. Cancer-associated fibroblasts (CAFs) promote epithelial–mesenchymal transition (EMT) and metastatic progression through extracellular matrix (ECM) remodeling and TGF-β signaling; however, the role of histamine …

jp (code pays fourni par la source)

0 citations Figshare
Profil ORCIDNotice OpenAlex

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